DNAase I Hypersensitive Site 3′ to the β-Globin Gene Cluster Containing Two TAA Insertions and a G→A Polymorphism is Predominantly Associated with the β⁺-Thalassemia IVS-I-6 (T→C) Mutation

Juliana T. N Martins Haematology and Haemotherapy Centre, State University of Campinas, Rua Carlos Chagas, 480, Cidade Universitária Zeferino Vaz, Barão Geraldo, Campinas, CEP: 13083-970, São Paulo, Brazil

Silvana Bordin Department of Physiology and Biophysics, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil

Dulcinéia M. de Albuquerque Haematology and Haemotherapy Centre, State University of Campinas, Rua Carlos Chagas, 480, Cidade Universitária Zeferino Vaz, Barão Geraldo, Campinas, CEP: 13083-970, São Paulo, Brazil

Sara T. O Saad Haematology and Haemotherapy Centre, State University of Campinas, Rua Carlos Chagas, 480, Cidade Universitária Zeferino Vaz, Barão Geraldo, Campinas, CEP: 13083-970, São Paulo, Brazil; Department of Clinical Medicine, State University of Campinas, São Paulo, Brazil

Fernando F. Costa Haematology and Haemotherapy Centre, State University of Campinas, Rua Carlos Chagas, 480, Cidade Universitária Zeferino Vaz, Barão Geraldo, Campinas, CEP: 13083-970, São Paulo, Brazil; Department of Physiology and Biophysics, Institute of Biomedical Sciences, University of São Paulo, São Paulo, Brazil; Haematology and Haemotherapy Centre, State University of Campinas, Rua Carlos Chagas, 480, Cidade Universitária Zeferino Vaz, Barão Geraldo, Campinas, CEP: 13083-970, São Paulo, Brazil

DNAase I Hypersensitive Site 3′ to the β-Globin Gene Cluster Containing Two TAA Insertions and a G→A Polymorphism is Predominantly Associated with the β⁺-Thalassemia IVS-I-6 (T→C) Mutation

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DNAase I Hypersensitive Site 3′ to the β-Globin Gene Cluster Containing Two TAA Insertions and a G→A Polymorphism is Predominantly Associated with the β+-Thalassemia IVS-I-6 (T→C) Mutation

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DNAase I Hypersensitive Site 3′ to the β-Globin Gene Cluster Containing Two TAA Insertions and a G→A Polymorphism is Predominantly Associated with the β⁺-Thalassemia IVS-I-6 (T→C) Mutation