Severe α-Thalassemia Intermedia Due to a Compound Heterozygosity for the Highly Unstable Hb Adana (HBA2: c.179G>A) and a Novel Codon 24 (HBA2: c.75T>A) Mutation

Dewi MegawatiThe Eijkman Institute for Molecular Biology JakartaIndonesia;Biomedical Sciences Faculty of Medicine, Universitas Indonesia JakartaIndonesia

Ita M. NainggolanThe Eijkman Institute for Molecular Biology JakartaIndonesia;Biomedical Sciences Faculty of Medicine, Universitas Indonesia JakartaIndonesia

Maria SwastikaThe Eijkman Institute for Molecular Biology JakartaIndonesia

Susi SusanahDepartment of Pediatric Hematogy-Oncology, Hasan Sadikin General Hospital/Faculty of Medicine Padjajaran University BandungIndonesia

Johanes C. MoseDepartment of Obstetric-Gynecology, Hasan Sadikin General Hospital/Faculty of Medicine Padjajaran University BandungIndonesia

Alida R. HarahapThe Eijkman Institute for Molecular Biology JakartaIndonesia;Department of Clinical Pathology, Faculty of Medicine, Universitas Indonesia JakartaIndonesia

Iswari SetianingsihThe Eijkman Institute for Molecular Biology JakartaIndonesiaCorrespondence[email protected]

show all

Severe α-Thalassemia Intermedia Due to a Compound Heterozygosity for the Highly Unstable Hb Adana (HBA2: c.179G>A) and a Novel Codon 24 (HBA2: c.75T>A) Mutation

Information for

Open access

Opportunities

Help and information

Your download is now in progress and you may close this window

Login or register to access this feature

Severe α-Thalassemia Intermedia Due to a Compound Heterozygosity for the Highly Unstable Hb Adana (HBA2: c.179G>A) and a Novel Codon 24 (HBA2: c.75T>A) Mutation

Reprints and Permissions

Related research

To cite this article:

Download citation

Information for

Open access

Opportunities

Help and information

Keep up to date