Interaction of an α⁺-Thalassemia Deletion with Either a Highly Unstable α-Globin Variant (α2, Codon 59, GGC→GAC) or a Nondeletional α-Thalassemia Mutation (AATAA→AATAAG): Comparison of Phenotypes Illustrating “Dominant” α-Thalassemia

J. Traeger-Synodinos First Department of Pediatrics, University of Athens St. Sophia's Children's Hospital, Athens, 11527, Greece

A. Metaxotou-Mavrommati First Department of Pediatrics, University of Athens St. Sophia's Children's Hospital, Athens, 11527, Greece

M. Karagiorga Thalassemia Unit, St. Sophia's Children's Hospital, Athens, 11527, Greece

C. Vrettou First Department of Pediatrics, University of Athens St. Sophia's Children's Hospital, Athens, 11527, Greece

I. Papassotiriou Hematology Laboratory, St. Sophia's Children's Hospital, Athens, 11527, Greece; Department of Clinical Biochemistry, St. Sophia's Children's Hospital, Athens, 11527, Greece

A. Stamoulakatou Hematology Laboratory, St. Sophia's Children's Hospital, Athens, 11527, Greece

E. Kanavakis First Department of Pediatrics, University of Athens St. Sophia's Children's Hospital, Athens, 11527, GreeceCorrespondence[email protected]

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Interaction of an α⁺-Thalassemia Deletion with Either a Highly Unstable α-Globin Variant (α2, Codon 59, GGC→GAC) or a Nondeletional α-Thalassemia Mutation (AATAA→AATAAG): Comparison of Phenotypes Illustrating “Dominant” α-Thalassemia

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Interaction of an α+-Thalassemia Deletion with Either a Highly Unstable α-Globin Variant (α2, Codon 59, GGC→GAC) or a Nondeletional α-Thalassemia Mutation (AATAA→AATAAG): Comparison of Phenotypes Illustrating “Dominant” α-Thalassemia

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Interaction of an α⁺-Thalassemia Deletion with Either a Highly Unstable α-Globin Variant (α2, Codon 59, GGC→GAC) or a Nondeletional α-Thalassemia Mutation (AATAA→AATAAG): Comparison of Phenotypes Illustrating “Dominant” α-Thalassemia