B cell TLR1/2, TLR4, TLR7 and TLR9 interact in induction of class switch DNA recombination: Modulation by BCR and CD40, and relevance to T-independent antibody responses

Egest J. PoneInstitute for Immunology, University of California, Irvine, CA, USA,

Zheng LouDepartment of Microbiology and Immunology, School of Medicine, University of Texas Health Science Center, San Antonio, TX, USA,

Tonika LamDepartment of Microbiology and Immunology, School of Medicine, University of Texas Health Science Center, San Antonio, TX, USA, ;Department of Molecular Biology & Biochemistry, University of California, Irvine, CA, USA,

Milton L. GreenbergDepartment of Physiology & Biophysics, University of California, Irvine, CA, USA, and

Rui WangDepartment of Microbiology and Immunology, School of Medicine, University of Texas Health Science Center, San Antonio, TX, USA, ;Xiangya Medical School, Central South University of China, Changsha, Hunan Province, China

Zhenming XuDepartment of Microbiology and Immunology, School of Medicine, University of Texas Health Science Center, San Antonio, TX, USA, Correspondence[email protected] [email protected]

Paolo CasaliInstitute for Immunology, University of California, Irvine, CA, USA, ;Department of Microbiology and Immunology, School of Medicine, University of Texas Health Science Center, San Antonio, TX, USA, Correspondence[email protected] [email protected]

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B cell TLR1/2, TLR4, TLR7 and TLR9 interact in induction of class switch DNA recombination: Modulation by BCR and CD40, and relevance to T-independent antibody responses

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B cell TLR1/2, TLR4, TLR7 and TLR9 interact in induction of class switch DNA recombination: Modulation by BCR and CD40, and relevance to T-independent antibody responses

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