Semliki Forest virus infection leads to increased expression of adhesion molecules on splenic T-cells and on brain vascular endothelium

Merja Soilu-hänninen Turku Immunology Centre University of Turku, Kiinamyllynkatu 13, FIN-20520, Turku, Finland; Department of Virology, University of Turku, Kiinamyllynkatu 13, FIN-20520, Turku, Finland

Matias Röyttä Turku Immunology Centre University of Turku, Kiinamyllynkatu 13, FIN-20520, Turku, Finland; Department of Pathology, University of Turku, Kiinamyllynkatu 13, FIN-20520, Turku, Finland

Aimo A. Salmi Turku Immunology Centre University of Turku, Kiinamyllynkatu 13, FIN-20520, Turku, Finland; Department of Virology, University of Turku, Kiinamyllynkatu 13, FIN-20520, Turku, Finland

Reijo Salonen Turku Immunology Centre University of Turku, Kiinamyllynkatu 13, FIN-20520, Turku, Finland; Department of Virology, University of Turku, Kiinamyllynkatu 13, FIN-20520, Turku, Finland; Glaxo-Wellcome, Ahventie 4, FIN-02170, Espoo, Finland

Semliki Forest virus infection leads to increased expression of adhesion molecules on splenic T-cells and on brain vascular endothelium

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Semliki Forest virus infection leads to increased expression of adhesion molecules on splenic T-cells and on brain vascular endothelium

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