References
- Hayatte S, Gaudoux M, Martel S, Bertrand S, Tigaud I, Magaud JP, et al. FLRG (follistatin-related gene), a new target of chromosomal rearrangement in malignant blood disorders. Oncogene 1998; 16: 2949–54
- Alan S, Amy S, Alicia Q, Yisrael S. Differential binding and neutralization of activin A and B by follistatin and follistatin like-3 (FSTL-3/FSRP/FLRG). Endocrinology 2003; 144: 1671–4
- Urbanek M, Lergo RS, Driscoll DA, Norman RJ. Thirty-seven candidate genes for polycystic ovary syndrome: strongest evidence for linkage is with follistatin. Proc Natl Acad Sci USA 1999; 96: 8573–8
- Xia Y, Sidis Y, Schneyer A. Overexpression of follistatin like-3 (FSTL3) in gonads causes defects in gonadal development and function in transgenic mice. Mol Endocrinol 2004; 18: 979–94
- Takebayashi K, Takakura K, Wang HQ, Kimura F, Kasahara K, Noda Y. Mutation analysis of the growth differentiation factor-9 and -9B genes in patients with premature ovarian failure and polycystic ovary syndrome. Fertil Steril 2000; 74: 976–9