Helicobacter pylori and Gastro-oesophageal Reflux Disease: Association and Clinical Implications: To Treat or Not to Treat with anti- H. pylori Therapy?

References

• Hulst RWM van der, Rauws EAJ, Koycu B, et al. Prevention of ulcer recurrence after successful eradication of Helicobacter pylori infection: a prospective long-term follow-up study. Gastroenterology 1997;113:1082–6.
   Google Scholar
• Labenz J, Blum AL, Bayerdorfer E, Meining A, Stolte M, Borsch G. Curing Helicobacte rpylori infectio n in patient s with duodenal ulcer may provoke reflux esophagitis. Gastroenterology 1997; 112:1442–7.
   PubMed Web of Science ®Google Scholar
• de Koster E, Kuipers EJ. Reflux and Helicobacter pylori. Curr Opin Gastroenterol 1997;13:43–7.
   Google Scholar
• Kuipers EJ, Klinkenberg-Knol EC, Meuwissen SG. Helicobacter pylori, proton pump inhibitors and gastroesophageal reflux disease. Yale J Biol Med 1999;72:211–8.
   PubMed Web of Science ®Google Scholar
• Calam J, Gibbons A, Healey ZV, Bliss P, Arebi N. How does Helicobacter pylori cause mucosal damage? Its effect on acid and gastrin physiology. Gastroenterology 1997;113:S43–9.
   Google Scholar
• Moss SF, Calam J. Acid secretion and sensitivity to gastrin in patients with duodenal ulcer: effect of eradication of Helicobacter pylori. Gut 1993;34:888–92.
   PubMed Web of Science ®Google Scholar
• El-Omar E, Penman I, Darrion CA, Ardhill JS, McColl KEL. Eradicating Helicobacte r pylori infection lowers gastrin mediated acid secretion by two thirds in patients with duodenal ulcer. Gut 1993;34: 1060–5.
   Google Scholar
• Beales IL. H. pylori-associated hypochlorhydria. Gastroenterology 1998;114:618–21.
   Google Scholar
• Porro GB, Pace F. Should we eradicate Helicobacter pylori in patients with recurrent gastro-oesophageal reflux disease. Eur J Gastroenterol Hepatol 2000;12 Suppl:7–10.
   Google Scholar
• Meyer-Rosberg K, Scott dR, Rex D, Melchers K, Sachs G. The effect of environmental pH on the protonmotive force of Helicobacter pylori. Gastroenterology 1996:111;886–900.
   Google Scholar
• Bell W, Birkholz C, Wagner S, Sewing KF. Interaction of Helicobacter pylori and its fatty acids with parietal cells and gastric H+/K+-ATP-ase. Gut 1994;35:1176–80.
   Google Scholar
• Kuipers EJ, Uyterlinde AM, Pena AS, Hazenberg HJ, Lindeman J, Klinkenber-Knol EC, et al. Increase of Helicobacter pylori associated corpus gastritis during acid suppressive therapy: implications for long-term safety. Am J Gastroenterol 1995;90: 1401–6.
   Google Scholar
• Werdmuller BFM, Loffeld RJLF. Helicobacter pylori and reflux esophagitis. Dig Dis Sci 1997;42:103–5.
   PubMed Web of Science ®Google Scholar
• Vicari JJ, Peek RM, Falk GW, Goldblum JR, Easley KA, Schnell J, et al. The seroprevalenc e of CagA-positive Helicobacte rpylori strains in the spectrum of gastroesophageal reflux disease. Gastroenterology 1998;115:50–7.
   PubMed Web of Science ®Google Scholar
• Varanasi RV, Fantry GT, Wilson KT. Decreased prevalence of Helicobacter pylori infection in gastroesophageal reflux disease. Helicobacter 1998;3:188–94.
   PubMed Web of Science ®Google Scholar
• Loffeld RJLF, Werdmuller BFM, Kuster JG, Perez-Perez GI, Blaser MJ, Kuipers EJ. Colonisation with cagA-positive Helicobacter pylori strains inversely associated with reflux esophagitis and Barrett’s esophagus. Digestion 2000;62:95–9.
   Google Scholar
• Vieth M, Massoud B, Meining A, Stolte M. Helicobacter pylori infection: protection against Barrett’s mucosa and neoplasia? Digestion 2000;62: 225–31.
   Google Scholar
• El-Seraq HB, Sonnenberg A, Jamal MM, Inadomi JM, Crooks LA, Feddersen RM. Corpus gastritis is protective against reflux oesophagitis. Gut 1999;45:181–5.
   Google Scholar
• Weston AP, Badr AS, Topalovski M, Cherian R, Dixon A, Hassanein RS. Prospective evaluation of the prevalence of gastric Helicobacter pylori infection in patients with GERD, Barrett’s esophagus, Barrett’s dysplasia, and Barrett’s adenocarcinoma. Am J Gastroenterol 2000;95:387–94.
   PubMedGoogle Scholar
• Schenk BE, Kuipers EJ, Klinkenberg-Knol EC, Eskes SA, Meuwissen SG. Helicobacter pylori and the efficacy of omeprazole therapy for gastroesophageal reflux. Am J Gastroenterol 1999;94:884–7.
   PubMedGoogle Scholar
• Holtmann G, Cain C, Malfertheiner P. Gastric Helicobacter pylori infection accelerates healing of reflux esophagitis during treatment with proton pump inhibitor pantoprazole. Gastroenterology 1999;117:11–6.
   Google Scholar
• Tefera S, Hatlebakk JG, Berstad A. The effect of Helicobacter pylori eradication on gastro-oesophageal reflux. Aliment Pharmacol Ther 1999;13:915–20.
   Google Scholar
• Sacca N, De Medici A, Rodino S, De Siena M, Giglio A. Reflux esophagitis: a complication of Helicobacter pylori eradication? Gut 1996;39 Suppl 2:A91.
   Google Scholar
• Hamada H, Haruma K, Mihara M, Kamada T, Yoshira M, Sumii K, et al. High incidence of reflux oesophagitis after eradication therapy for Helicobacter pylori: impacts of hiatal hernia and corpus gastritis. Aliment Pharmacol Ther 2000;14:729–35.
   PubMed Web of Science ®Google Scholar
• Fallone C, Barkun AN, Friedman G, Mayrand S, Loo V, Beech R, et al. Is Helicobacter pylori eradication associated with gastroesophageal reflux disease? Am J Gastroenterol 2000;95: 914–20.
   PubMed Web of Science ®Google Scholar
• Martinek J, Kuzela L, Spicak J, Vavrecka A. The clinical influence of Helicobacter pylori in effective acid suppression; implications for the treatment of gastro-oesophageal reflux disease. Aliment Pharmacol Ther 2000;14:979–90.
   Google Scholar
• Verdu EF, Armstrong D, Idstrom JP, Labenz J, Stolte M, Borsch G, et al. Intragastric pH during treatment with omeprazole: role of Helicobacter pylori and H. pylori-associated gastritis. Scand J Gastroenterol 1996;31:1151–6.
   PubMed Web of Science ®Google Scholar
• Labenz J, Tillenburg B, Peitz U, Idstrom JP, Verdu EF, Stolte M, et al. Helicobacter pylori augments the pH-increasing effect of omeprazole in patients with duodenal ulcer. Gastroenterology 1996;110:735–32.
   Google Scholar