Amantadine for neurobehavioural deficits following delayed post-hypoxic encephalopathy

References

• CHoi, S. I.: Delayed neurologic sequelae in carbon monoxide intoxication. Archives of Neurology, 40: 433–435, 1983.
   PubMed Web of Science ®Google Scholar
• PLUM, F., POSNER, J. and HAIN, R.: Delayed neurological deterioration after anoxia. Archives of Internal Medicine, 110: 56–63, 1962.
   Google Scholar
• GINSBERG, M. D.: Delayed neurological deterioration following hypoxia. Neurology, 26: 21–44, 1979.
   Google Scholar
• SHILLITO, F. H., DRINKER, C. K. and SHAUGHNESSY, T. J.: The problem of nervous and mental sequelae in carbon monoxide poisoning. Journal of American Medical Association, 106: 669–674, 1936.
   Google Scholar
• LEE, M. H.: Clinical studies on delayed sequelae of carbon monoxide intoxication. Journal of Korean Neuropsychiatry Association, 15: 374–385, 1978.
   Google Scholar
• GOTTFRIED, J. A., MAYER, S. A., SHUNGU, D. et al.: Delayed posthypoxic demyelination: associa-tion with arylsulfatase A deficiency and lactic acidosis on proton MR spectroscopy. Neurology, 49: 1400–1404, 1997.
   PubMed Web of Science ®Google Scholar
• WEINBERGER, L. M., SCHMIDLEY, J. W., SCHAFTER, I. A. et al.: Delayed posthypoxic demyelination and arylsulfatase A pseudodeficiency. Neurology, 44: 152–154, 1994.
   PubMed Web of Science ®Google Scholar
• DEUTSCH, H.: Ein fall symmetrischer erweichung im streifenhugel und im linsenkem. Jahrbucher Psychiatry Neurology, 37: 237–254, 1917.
   Google Scholar
• PROTASS, L.: Delayed postanoxic encephalopathy after heroin use. Annals of Internal Medicine, 74: 738–739, 1971.
   PubMed Web of Science ®Google Scholar
• COURVILLE, C. B.: Case studies in cerebral anoxia. V. Characteristic anoxic alterations in cortex and corpus striatrum consequent to strangulation. Bulletin of Los Angeles Neurologic Society, 20: 9–19, 1955.
   Google Scholar
• RICHARDSON, J. C., CHAMBERS, R. A. and HEYWOOD, P. M.: Encephalopathies of anoxia and hyperglycemia. Archives of Neurology, 1: 178–190, 1959.
   Web of Science ®Google Scholar
• GAMPER, E. and STIEFLER, G.: Klinisches bild und anatomischer befund nach drosselung. Archly for Psychiatrie Nervenkrankheiten, 106: 774–778, 1937.
   Google Scholar
• DOOLING, E. C. and RICHARDSON, E. P. JR.: Delayed encephalopathy after strangling. Archives of Neurology, 33: 196–199, 1976.
   PubMedGoogle Scholar
• MERIWETHER, L. S., HAGER, H. and ScHoLz, W.: Cerebral pathology following serum anaphy-laxis. Archives of Neurology and Psychiatry, 73: 286–292, 1955.
   Google Scholar
• MEIGS, J. W. and HUGHES, J. P. W.: Acute carbon monoxide poisoning. Archives of Industrial Hygiene, 6: 344–356, 1939.
   Google Scholar
• GRINICER, R.: Parkinsonism following carbon monoxide poisoning. Journal of Nervous and Mental Disorders, 64: 18–28, 1926.
   Google Scholar
• MEYTHALER, J. M., BRUNNER, R. C., JOHNSON, A. et al.: Amantadine to improve neurorecovery in traumatic brain injury-associated diffuse axonal injury: a pilot double-blind randomized trial. Journal of Head Trauma Rehabilitation, 17: 300–313, 2002.
   PubMed Web of Science ®Google Scholar
• GUALTIERI, T., CHANDLER, M., COONS, T. B. et al.: Amantadine: a new clinical profile for traumatic brain injury. Clinical Neuropharmacology, 12: 258–270, 1989.
   PubMed Web of Science ®Google Scholar
• MARIN, R. S., FOGEL, B. S., HAWICINS, J. et al.: Apathy: a treatable syndrome. Journal of Neuropsychiatry, 7: 23–30, 1995.
   PubMed Web of Science ®Google Scholar
• KARL', D. C., BURKE, D. T., KIM, H. J. et al.: Effects of dopaminergic combination therapy for frontal lobe dysfunction in traumatic brain injury rehabilitation. Brain Injury, 13: 63–68, 1999.
   PubMed Web of Science ®Google Scholar
• SOHLBERG, M. and MATEER, C.: Attention Process Training (APT) (Puyallup Washington: Association for Neuropsychological Research and Development), 1986.
   Google Scholar
• WILSON, B., COCICBURN, J. and BADDELEY, A.: Rivermead Behavioural Memory Test (Titchfield, UK: Thames Valley Test Company), 1989.
   Google Scholar
• COURVILLE, C. B.: The process of demyelination in the central nervous system: IV. Demyelination as a delayed residual of carbon monoxide asphyxia. Journal of Nervous and Mental Disorders, 125: 534–546, 1957.
   Google Scholar
• SCHWEDENBERG, T. H.: Leukoencephalopathy following carbon monoxide asphyxia. Journal of Neuropathological Experiments in Neurology, 18: 597–608, 1959.
   PubMed Web of Science ®Google Scholar
• GINSBERG, M. D. and MYERS, R.: Experimental carbon monoxide encephalopathy in the primate. Physiologic and metabolic aspects. Archives of Neurology, 30: 202–208, 1974.
   Google Scholar
• BARKER, R. A., DICK, D. J. and CocHius, J. I.: Delayed postanoxic encephalopathy and pseudodeficiency of arylsulfatase A. Journal of Neurology, 246: 1103–1104, 1999.
   PubMed Web of Science ®Google Scholar
• MEYER, J. E.: Himvaranderungen vom Typ det CO-Vergiftung nach Stickoxydul-Ather-Narkose. Archives of Psychiatry Nervenkr, 202: 113–125, 1961.
   Google Scholar
• COURVILLE, C. B.: Contributions to the study of cerebral anoxia (Los Angeles: San Lucas Press), 1953.
   Google Scholar
• GINSBERG, M. D., HEDLEY-WHYTE, T. and RICHARDSON, E. P. JR.: Hypoxic-ischemic leuko-encephalopathy in man. Archives of Neurology, 33: 5–14, 1976.
   Google Scholar
• ULE, G.: Uber eine Todliche, klinisch intervallare Nartiumnitrat-Vergiftung. Arztliche Wochenschrift, 13: 1038–1040, 1958.
   PubMedGoogle Scholar
• MEGA, M. S. and CUMMINGS, J. L.: Frontal-subcortical circuits and neuropsychiatric disorders. Journal of Neuropsychiatry and Clinical Neuroscience, 6: 358–370, 1994.
   PubMed Web of Science ®Google Scholar