Pro-inflammatory cytokines as potential predictors for intradialytic hypotension

References

• Chou Jason A, Kalantar-Zadeh K, Mathew AT. A brief review of intradialytic hypotension with a focus on survival. Semin Dial. 2017;30(6):473–480.
   PubMed Web of Science ®Google Scholar
• Flythe JE, Xue H, Lynch KE, et al. Association of mortality risk with various definitions of intradialytic hypotension. JASN. 2015;26(3):724–734.
   Web of Science ®Google Scholar
• Yu J, Liu Z, Shen B, et al. Intradialytic hypotension as an independent risk factor for long-term mortality in maintaining hemodialysis patients: a 5-year follow-up cohort study. Blood Purif. 2018;45(4):320–326.
   PubMed Web of Science ®Google Scholar
• Stefansson BV, Brunelli SM, Cabrera C, et al. Intradialytic hypotension and risk of cardiovascular disease. Clin J Am Soc Nephrol. 2014;9(12):2124–2132.
   PubMed Web of Science ®Google Scholar
• Wu T-K, Lim P-S, Jin J-S, et al. Impaired gut epithelial tight junction expression in hemodialysis patients complicated with intradialytic hypotension. Biomed Res Int. 2018;2018:1–6.
   Web of Science ®Google Scholar
• MacEwen C, Sutherland S, Daly J, et al. Relationship between hypotension and cerebral ischemia during hemodialysis. J Am Soc Nephrol. 2017;28(8):2511–2520.
   PubMed Web of Science ®Google Scholar
• Yuen D, Chan CT. Inflammation, cardiovascular disease and nocturnal hemodialysis. Curr Opin Nephrol Hypertens. 2005;14(6):538–542.
   PubMed Web of Science ®Google Scholar
• Kalantar-Zadeh K, Block G, McAllister CJ, et al. Appetite and inflammation, nutrition, anemia, and clinical outcome in hemodialysis patients. Am J Clin Nutr. 2004;80(2):299–307.
   PubMed Web of Science ®Google Scholar
• Kaysen GA, Greene T, Daugirdas JT, et al.; HEMO Study Group. Longitudinal and cross-sectional effects of C-reactive protein, equilibrated normalized protein catabolic rate, and serum bicarbonate on creatinine and albumin levels in dialysis patients. Am J Kidney Dis. 2003;42(6):1200–1211.
   PubMed Web of Science ®Google Scholar
• Jones CH, Akbani H, Croft DC, et al. The relationship between serum albumin and hydration status in hemodialysis patients. J Ren Nutr. 2002;12(4):209–212.
   PubMed Web of Science ®Google Scholar
• McIntyre CW, Harrison LEA, Eldehni MT, et al. Circulating endotoxemia: a novel factor in systemic inflammation and cardiovascular disease in chronic kidney disease. Clin J Am Soc Nephrol. 2011;6(1):133–141.
   PubMed Web of Science ®Google Scholar
• Pertosa G, Grandaliano G, Gesualdo L, et al. Clinical relevance of cytokine production in hemodialysis. Kidney Int Suppl. 2000;76:S104–S11.
   PubMedGoogle Scholar
• Ding X. SIKD is on the road. Blood Purif. 2016;42(1):I–VI.
   PubMed Web of Science ®Google Scholar
• Cao X-S, Chen J, Zou J-Z, et al. Association of indoxyl sulfate with heart failure among patients on hemodialysis. Clin J Am Soc Nephrol. 2015;10(1):111–119.
   PubMed Web of Science ®Google Scholar
• K/DOQI Workgroup. K/DOQI clinical practice guidelines for cardiovascular disease in dialysis patients. Am J Kidney Dis. 2005;45:S1–S153.
   Web of Science ®Google Scholar
• Chou JA, Streja E, Nguyen DV, et al. Intradialytic hypotension, blood pressure changes and mortality risk in incident hemodialysis patients. Nephrol Dialysis Transpl. 2018;33(1):149–159.
   PubMed Web of Science ®Google Scholar
• Assimon MM, Flythe JE. Intradialytic blood pressure abnormalities: the highs, the lows and all that lies between. Am J Nephrol. 2015;42(5):337–350.
   PubMed Web of Science ®Google Scholar
• Daugirdas JT. Pathophysiology of dialysis hypotension: an update. American journal of kidney diseases: the official journal of the National Kidney Foundation. Am J Kidney Dis. 2001;38(4 Suppl 4):S11–S17.
   PubMed Web of Science ®Google Scholar
• Oberg BP, McMenamin E, Lucas FL, et al. Increased prevalence of oxidant stress and inflammation in patients with moderate to severe chronic kidney disease. Kidney Int. 2004;65(3):1009–1016.
   PubMed Web of Science ®Google Scholar
• Shlipak MG, Fried LF, Crump C, et al. Elevations of inflammatory and procoagulant biomarkers in elderly persons with renal insufficiency. Circulation. 2003;107(1):87–92.
   PubMed Web of Science ®Google Scholar
• Pecoits-Filho R, Heimbürger O, Bárány Peter Suliman M, et al. Associations between circulating inflammatory markers and residual renal function in CRF patients. Am J Kidney Dis. 2003;41(6):1212–1218.
   PubMed Web of Science ®Google Scholar
• Pereira BJ, Shapiro L, King AJ, et al. Plasma levels of IL-1 beta, TNF alpha and their specific inhibitors in undialyzed chronic renal failure, CAPD and hemodialysis patients. Kidney Int. 1994;45(3):890–896.
   PubMed Web of Science ®Google Scholar
• Dekker MJE, Marcelli D, Canaud BJ, et al.; MONDO Initiative. Impact of fluid status and inflammation and their interaction on survival: a study in an international hemodialysis patient cohort. Kidney Int. 2017;91(5):1214–1223.
   PubMed Web of Science ®Google Scholar
• Hassan MO, Duarte R, Dix-Peek T, et al. Correlation between volume overload, chronic inflammation, and left ventricular dysfunction in chronic kidney disease patients. CN. 2016;86(S1):131–135.
   Google Scholar
• Dekker MJE, van der Sande FM, van den Berghe F, et al. Fluid overload and inflammation axis. Blood Purif. 2018;45(1-3):159–165.
   PubMed Web of Science ®Google Scholar
• John B, Tan BK, Dainty S, et al. Plasma volume, albumin, and fluid status in peritoneal dialysis patients. CJASN. 2010;5(8):1463–1470.
   PubMed Web of Science ®Google Scholar
• Jones CH, Wells L, Stoves J, et al. Can a reduction in extracellular fluid volume result in increased serum albumin in peritoneal dialysis patients. Am J Kidney Dis. 2002;39(4):872–875.
   PubMed Web of Science ®Google Scholar
• Kooman JP, Dekker MJ, Usvyat LA, et al. Inflammation and premature aging in advanced chronic kidney disease. Am J Physiol Renal Physiol. 2017;313(4):F938–F950.
   PubMed Web of Science ®Google Scholar
• Lau WL, Kalantar-Zadeh K, Vaziri ND. The gut as a source of inflammation in chronic kidney disease. Nephron. 2015;130(2):92–98.
   PubMed Web of Science ®Google Scholar
• Erkan E, Devarajan P, Kaskel F. Role of nitric oxide, endothelin-1, and inflammatory cytokines in blood pressure regulation in hemodialysis patients. Am. J. Kidney Dis. 2002;40(1):76–81.
   PubMed Web of Science ®Google Scholar
• Ouviña SM, La Greca RD, Zanaro NL, et al. Endothelial dysfunction, nitric oxide and platelet activation in hypertensive and diabetic type II patients. Thromb Res. 2001;102(2):107–114.
   PubMed Web of Science ®Google Scholar
• Bhagat K, Vallance P. Inflammatory cytokines impair endothelium-dependent dilatation in human veins in vivo. Circulation. 1997;96(9):3042–3047.
   PubMed Web of Science ®Google Scholar
• Dinarello CA, Koch KM, Shaldon S. Interleukin-1 and its relevance in patients treated with hemodialysis. Kidney Int Suppl. 1988;24:S21–S6.
   PubMedGoogle Scholar
• Yoshizumi M, Perrella M, Burnett JC, et al. Tumor necrosis factor downregulates an endothelial nitric oxide synthase mRNA by shortening its half-life. Circ Res. 1993;73(1):205–209.
   PubMed Web of Science ®Google Scholar
• Kang ES, Acchiardo SR, Kang AH. Implications for the role of endogenous nitric oxide inhibitors in hemodialysis hypotension. Free Radic Res. 2001;35(4):341–365.
   PubMed Web of Science ®Google Scholar
• Beerenhout CH, Noris Marina Kooman JP, Porrati F, et al. Nitric oxide synthetic capacity in relation to dialysate temperature. Blood Purif. 2004;22(2):203–209.
   PubMed Web of Science ®Google Scholar
• Schneditz D, Rosales L, Kaufman AM, et al. Heat accumulation with relative blood volume decrease. Am J Kidney Dis. 2002;40(4):777–782.
   PubMed Web of Science ®Google Scholar
• van der Sande FM, Kooman JP, Burema JH, et al. Effect of dialysate temperature on energy balance during hemodialysis: quantification of extracorporeal energy transfer. Am J Kidney Dis. 1999;33(6):1115–1121.
   PubMed Web of Science ®Google Scholar
• Tomita M, Malhotra D, Dheenan S, et al. A potential role for immune activation in hemodialysis hypotension. Ren Fail. 2001;23(5):637–649.
   PubMed Web of Science ®Google Scholar
• McIntyre CW, Burton JO, Selby NM, et al. Hemodialysis-induced cardiac dysfunction is associated with an acute reduction in global and segmental myocardial blood flow. Clin J Am Soc Nephrol. 2008;3(1):19–26.
   PubMed Web of Science ®Google Scholar
• Burton JO, Jefferies HJ, Selby NM, et al. Hemodialysis-induced cardiac injury: determinants and associated outcomes. CJASN. 2009;4(5):914–920.
   PubMed Web of Science ®Google Scholar
• Diebel L, Kozol R, Wilson RF, et al. Gastric intramucosal acidosis in patients with chronic kidney failure. Surgery. 1993;113(5):520–526.
   PubMed Web of Science ®Google Scholar
• Yu AW, Nawab ZM, Barnes WE, et al. Splanchnic erythrocyte content decreases during hemodialysis: a new compensatory mechanism for hypovolemia. Kidney Int. 1997;51(6):1986–1990.
   PubMed Web of Science ®Google Scholar
• Khanna A, Rossman JE, Fung HL, et al. Intestinal and hemodynamic impairment following mesenteric ischemia/reperfusion. J Surg Res. 2001;99(1):114–119.
   PubMed Web of Science ®Google Scholar