18
Views
2
CrossRef citations to date
0
Altmetric
Original Article

Expression of the Candidate A-T Gene ATDC Is Not Detectable in a Human Cell Line with a Normal Response to Ionizing Radiation

, , &
Pages S77-S84 | Received 23 May 1994, Accepted 24 Jun 1994, Published online: 03 Jul 2009

References

  • Abate C., Curran T. Encounters with Fos and Jun on the road to AP-1. Seminars in Cancer Biology 1990; 1: 19–26
  • Busch S.J., Sassone-Corsi P. Dimers, leucine zippers and DNA-binding domains. Trends in Genetics 1990; 6: 36–40
  • Fields S., Song O. A novel genetic system to detect protein-protein interactions. Nature 1989; 340: 245–246
  • Gatti R.A., Boder E., Vinters H.V., Sparkes R.S., Norman A., Lange K. Ataxiatelangiectasia: an interdisciplinary approach to pathogenesis. Medicine 1991; 70: 99–117
  • Grignani F., Ferrucci P.F., Testa U., Talamo G., Fagioli M., Alcalay M., Mencarelli A., Grignani F., Peschle C., Nicoletti I., Pelicci P.G. The acute promyelocytic leukemia-specific PML-RARα fusion protein inhibits differentiation and promotes survival of myeloid precursor cells. Cell 1993; 74: 423–431
  • Gyuris J., Golemis E., Chertkov H., Brent R. Cdil, a human G1 and S phase protein phosphatase that associates with Cdk2. Cell 1993; 75: 791–803
  • Hardy C.F.J., Sussel L., Shore D. A RAP1-interacting protein involved in transcriptional silencing and telomere length regulation. Genes and Development 1992; 6: 801–814
  • Herskowitz I. Functional inactivation of genes by dominant negative mutations. Nature 1987; 329: 219–222
  • Kakizuka A., Miller W.H., Jr., Umesono K., Warrell R.P., Jr., Frankel S.R., Murty V.V., Dmitrovsky E., Evans R.M. Chromosomal translocation t(15;17) in human acute promyelocytic leukemia fuses RARα with a novel putative transcription factor, PML. Cell 1991; 66: 663–674
  • Kapp L.N., Painter R.B. Stable radioresistance in ataxia-telangiectasia cells containing DNA from normal human cells. International Journal of Radiation Biology 1989; 56: 667–675
  • Kapp L.N., Painter R.B., Yu L.C., van Loon N., Richard C.W., III, James M.R., Cox D.R., Murnane J.P. Cloning of a candidate gene for ataxia-telangiectasia group D. American Journal of Human Genetics 1992; 51: 45–54
  • Kastan M.B., Zahn Q., El-Deiry W.S., Carrier F., Jacks T., Walsh W.V., Plunkett B.S., Vogelstein B., Fornace A.J., Jr. A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasia. Cell 1992; 71: 587–597
  • Kastner P., Perez A., Lutz Y., Rochette-Egly C., Gaub M.-P., Durand B., Lanotte M., Berger R., Chambon P. Structure, localization and transcriptional properties of two classes of retinoic acid receptor α fusion proteins in acute promyelocytic leukemia (APL): structural similarities with a new family of oncoproteins. EMBO Journal 1992; 11: 629–642
  • Kolodziej P.A., Young R.A. Epitope tagging and protein surveillance. Methods in Enzymology 1991; 194: 508–519
  • Kuerbitz S.J., Plunkett B.S., Walsh W.V., Kastan M.B. Wild-type p53 is a cell cycle checkpoint determinant following irradiation. Proceedings of the National Academy of Sciences, USA 1992; 89: 7491–7495
  • Lange E., Gatti R.A., Sobel E., Concannon P., Lange K. How many ataxia-telangiectasia genes?. Ataxia-telangiectasia, R.A. Gatti, R.B. Painter. Springer, Berlin 1993; 37–54, In
  • Leonhardt E.A., Kapp L.N., Young B.R., Murnane J.P. Nucleotide sequence analysis of a candidate gene for ataxia-telangiectasia group D (ATDC). Genomics 1994; 19: 130–136
  • Meyn M.S., Lu-Kuo J.M., Herzing L.B.K. Expression cloning of multiple human cDNAs that complement the phenotypic defects of ataxia-telangiectasia group D fibroblasts. American Journal of Human Genetics 1993; 53: 1206–1216
  • Miller J.H. Experiments in Molecular Genetics. Cold Spring Harbor Laboratory, Cold Spring Harbor Press. 1992
  • Murnane J.P., Kapp L.N. A critical look at the association of human genetic syndromes with sensitivity to ionizing radiation. Seminars in Cancer Biology 1993; 4: 93–104
  • Murnane J.P., Fuller L.F., Painter R.B. Establishment and characterization of a permanent pSVori--transformed ataxia-telangiectasia cell line. Experimental Cell Research 1985; 158: 119–126
  • Painter R.B., Young B.R. Radiosensitivity in ataxia-telangiectasia: a new explanation. Proceedings of the National Academy of Sciences, USA 1980; 77: 7315–7317
  • Ransone L.J., Visvader J., Wamsley P., Verma I.M. Trans-dominant negative mutants of Fos and Jun. Proceedings of the National Academy of Sciences, USA 1990; 87: 3806–3810
  • Sambrook J., Fritsch E.F., Maniatis T. Molecular Cloning: a Laboratory Manual2nd edn. Cold Spring Harbor Laboratory, Cold Spring Harbor Press. 1989
  • Sobel E., Lange E., Jaspers N.G.J., Chessa L., Sanal O., Shiloh Y., Taylor A.M.R., Weemaes C.M., Lange K., Gatti R.A. Ataxiatelangiectasia: linkage evidence for genetic heterogeneity [letter; comment]. American Journal of Human Genetics 1992; 50: 1343–1348
  • Taylor A.M.R., Byrd P.J., McConville C.M., Thacker S. Genetic and cellular features of ataxia-telangiectasia. International Journal of Radiation Biology 1994; 65: 65–70
  • van den Heuval S., Harlow E. Distinct roles for cyclin-dependent kinases in cell cycle control. Science 1993; 262: 2050–2054
  • Wigler M., Pellicer A., Silverstein S., Axel R. Biochemical transfer of single-copy eucaryotic genes using total cellular DNA as donor. Cell 1978; 14: 725–731
  • Yang X., Hubbard E.J., Carlson M. A protein kinase substrate identified by the two-hybrid system. Science 1992; 257: 680–682

Reprints and Corporate Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

To request a reprint or corporate permissions for this article, please click on the relevant link below:

Academic Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

Obtain permissions instantly via Rightslink by clicking on the button below:

If you are unable to obtain permissions via Rightslink, please complete and submit this Permissions form. For more information, please visit our Permissions help page.