199
Views
4
CrossRef citations to date
0
Altmetric
Articles

Gallic acid as a key substance to inhibit proliferation and adipogenesis in bovine subcutaneous adipocyte

ORCID Icon, ORCID Icon, ORCID Icon, ORCID Icon, ORCID Icon & ORCID Icon

References

  • Klaus S. Adipose tissue as a regulator of energy balance. Curr Drug Target. 2004;5(3):241–250.
  • Khan MI, Jo C, Tariq MR. Meat flavor precursors and factors influencing flavor precursors-A systematic review. Meat Sci. 2015;110:278–284.
  • National Academies of Sciences, Engineering, and Medicine. Nutrient requirements of beef cattle: Eighth revised edition. Washington, DC: The National Academies Press; 2016: 187.
  • Pesonen M, Huuskonen AK. Production, carcass characteristics and valuable cuts of beef breed bulls and heifers in Finnish beef cattle population. AFSci. 2015;24(3):164–172.
  • Kessler A, Kalske A. Plant secondary metabolite diversity and species interactions. Annu Rev Ecol Evol Syst. 2018;49(1):115–138.
  • Piluzza G, Sulas L, Bullitta S. Tannins in forage plants and their role in animal husbandry and environmental sustainability: a review. Grass Forage Sci. 2014;69(1):32–48.
  • Urbatzka R, Freitas S, Palmeira A, et al. Lipid reducing activity and toxicity profiles of a library of polyphenol derivatives. Eur J Med Chem. 2018;151:272–284.
  • Doan KV, Ko CM, Kinyua AW, et al. Gallic acid regulates body weight and glucose homeostasis through AMPK activation. Endocrinology. 2015;156(1):157–168.
  • Huang DW, Chang WC, Yang HJ, et al. Gallic acid alleviates hypertriglyceridemia and fat accumulation via modulating glycolysis and lipolysis pathways in perirenal adipose tissues of rats fed a high-fructose diet. IJMS. 2018;19(1):254.
  • Liu XM, Zhao HB, Jin Q, et al. Resveratrol induces apoptosis and inhibits adipogenesis by stimulating the SIRT1-AMPKα-FOXO1 signalling pathway in bovine intramuscular adipocytes. Mol Cell Biochem. 2018;439(1–2):213–223.
  • Hsu CL, Lo WH, Yen GC. Gallic acid induces apoptosis in 3T3-L1 pre-adipocytes via a Fas- and mitochondrial-mediated pathway. J Agric Food Chem. 2007;55(18):7359–7365.
  • Livak KJ, Schmittgen TD. Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) method. Methods. 2001;25(4):402–408.
  • Hsu CL, Huang SL, Yen GC. Inhibitory effect of phenolic acids on the proliferation of 3T3-L1 preadipocytes in relation to their antioxidant activity. J Agric Food Chem. 2006;54(12):4191–4197.
  • Shell SA, Lyass L, Trusk PB, et al. Activation of AMPK is necessary for killing cancer cells and sparing cardiac cells. Cell Cycle. 2008;7(12):1769–1775.
  • Ravinayagam V, Shehzad A, Almohazey D, et al. Decursin induces apoptosis by regulating AMP-activated protein kinase and Bax/Bcl-2 pathway in HepG2 cell line. Eur J Integr Med. 2018;24:17–22.
  • Hsu CL, Yen GC. Effects of flavonoids and phenolic acids on the inhibition of adipogenesis in 3T3-L1 adipocytes. J Agric Food Chem. 2007;55(21):8404–8410.
  • Wu L, Zhang L, Li B, et al. AMP-activated protein kinase (AMPK) regulates energy metabolism through modulating thermogenesis in adipose tissue. Front Physiol. 2018;9:122.
  • Beylot M. Metabolism of white adipose tissue. Adipose tissue and adipokines in health and disease. Totowa, NJ: Humana Press; 2014: 33–52.
  • Jiang D, Wang D, Zhuang X, et al. Berberine increases adipose triglyceride lipase in 3T3–L1 adipocytes through the AMPK pathway. Lipids Health Dis. 2016;15(1):214.

Reprints and Corporate Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

To request a reprint or corporate permissions for this article, please click on the relevant link below:

Academic Permissions

Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?

Obtain permissions instantly via Rightslink by clicking on the button below:

If you are unable to obtain permissions via Rightslink, please complete and submit this Permissions form. For more information, please visit our Permissions help page.