Up-regulation of serum amyloid A3 mRNA expression after stimulation with lipopolysaccharide and lipoteichoic acid in mouse lung cells

Yasuo InoshimaCooperative Department of Veterinary Medicine, Laboratory of Food and Environmental Hygiene, Gifu University, Gifu, Japan; ;Education and Research Center for Food Animal Health (GeFAH), Gifu University, Gifu, Japan; ;The United Graduate School of Veterinary Sciences, Gifu University, Gifu, Japan Correspondence[email protected]

http://orcid.org/0000-0002-1189-8392 View further author information

Ami IwataCooperative Department of Veterinary Medicine, Laboratory of Food and Environmental Hygiene, Gifu University, Gifu, Japan; View further author information

Ayaka OkadaCooperative Department of Veterinary Medicine, Laboratory of Food and Environmental Hygiene, Gifu University, Gifu, Japan; ;Education and Research Center for Food Animal Health (GeFAH), Gifu University, Gifu, Japan; View further author information

References

Reigstad CS, Lunden GÖ, Felin J, et al. Regulation of serum amyloid A3 (SAA3) in mouse colonic epithelium and adipose tissue by the intestinal microbiota. PLoS One. 2009;4:e5842.
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Shigemura H, Ishiguro N, Inoshima Y. Up-regulation of MUC2 mucin expression by serum amyloid A3 protein in mouse colonic epithelial cells. J Vet Med Sci. 2014;76:985–991.
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Tashiro M, Iwata A, Yamauchi M, et al. The N-terminal region of serum amyloid A3 protein activates NF-κB and up-regulates MUC2 mucin mRNA expression in mouse colonic epithelial cells. PLoS One. 2017;12:e0181796.
PubMed Web of Science ®Google Scholar

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Up-regulation of serum amyloid A3 mRNA expression after stimulation with lipopolysaccharide and lipoteichoic acid in mouse lung cells

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Up-regulation of serum amyloid A3 mRNA expression after stimulation with lipopolysaccharide and lipoteichoic acid in mouse lung cells

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