References
- Iwaszko M, Swierkot J, Kolossa K, et al. Influence of CD94 and NKG2A variants on susceptibility to rheumatoid arthritis and efficacy of anti-TNF treatment. Joint Bone Spine. 2016;83:75–79.
- Iwaszko M, Świerkot J, Kolossa K, et al. Polymorphisms within the human leucocyte antigen-E gene and their associations with susceptibility to rheumatoid arthritis as well as clinical outcome of anti-tumour necrosis factor therapy. Clin Exp Immunol. 2015;182:270–277.
- Emery P, Kavanaugh A, Bao Y, et al. Comprehensive disease control (CDC): what does achieving CDC mean for patients with rheumatoid arthritis? Ann Rheum Dis. 2015;74:2165–2174.
- Kharlamova N, Jiang X, Sherina N, et al. Antibodies to Porphyromonas gingivalis indicate interaction between oral infection, smoking and risk genes in rheumatoid arthritis etiology. Arthritis Rheumatol. 2016;68:604–613.
- Castro-Santos P, Diaz-Pena R. Genetics of rheumatoid arthritis: a new boost is needed in Latin American populations. Rev Bras Reumatol. 2016;56:171–177.
- Ruyssen-Witrand A, Lukas C, Nigon D, et al. Association of IL-2RA and IL-2RB genes with erosive status in early rheumatoid arthritis patients (ESPOIR and RMP cohorts). Joint Bone Spine. 2014;81:228–234.
- Combe B, Rincheval N. Early lessons from the recent-onset rheumatoid arthritis cohort ESPOIR. Joint Bone Spine. 2015;82:13–17.
- Spurlock CF, 3rd, Tossberg JT, Olsen NJ, et al. Cutting edge: chronic NF-kappaB activation in CD4+ T cells in rheumatoid arthritis is genetically determined by HLA risk alleles. JI. 2015;195:791–795.
- Lee YH, Bae SC. Association between susceptibility to rheumatoid arthritis and PADI4 polymorphisms: a meta-analysis. Clin Rheumatol. 2016;35:961–971.
- Ates A, Karaaslan Y, Karatayli E, et al. Association of the PTPN22 gene polymorphism with autoantibody positivity in Turkish rheumatoid arthritis patients. Tissue Antigens. 2011;78:56–59.
- Moens E, Brouwer M, Dimova T, et al. IL-23R and TCR signaling drives the generation of neonatal Vgamma9Vdelta2 T cells expressing high levels of cytotoxic mediators and producing IFN-gamma and IL-17. J Leukoc Biol. 2011;89:743–752.
- Kave M, Shadman M, Alizadeh A, et al. Analysis of the association between IL-23R rs11209026 polymorphism and incidence of atherosclerosis. Int J Immunogenet. 2015;42:341–345.
- Eken A, Singh AK, Treuting PM, et al. IL-23R + innate lymphoid cells induce colitis via interleukin-22-dependent mechanism. Mucosal Immunol. 2014;7:143–154.
- Wendling D, Cedoz JP, Racadot E. Serum and synovial fluid levels of p40 IL12/23 in spondyloarthropathy patients. Clin Rheumatol. 2009;28:187–190.
- Steinman L. A brief history of T(H)17, the first major revision in the T(H)1/T(H)2 hypothesis of T cell-mediated tissue damage. Nat Med. 2007;13:139–145.
- Cargill M, Schrodi SJ, Chang M, et al. A large-scale genetic association study confirms IL12B and leads to the identification of IL23R as psoriasis-risk genes. Am J Hum Genet. 2007;80:273–290.
- Nunez C, Dema B, Cenit MC, et al. IL23R: a susceptibility locus for celiac disease and multiple sclerosis?. Genes Immun. 2008; 9:289–293.
- Sato K, Suematsu A, Okamoto K, et al. Th17 functions as an osteoclastogenic helper T cell subset that links T cell activation and bone destruction. J Exp Med. 2006;203:2673–2682.
- Varade J, Ramon Lamas J, Rodriguez L, et al. IL23R and IL12B genes: susceptibility analysis in rheumatoid arthritis. Ann Rheum Dis. 2009;68:1230–1232.
- Hollis-Moffatt JE, Merriman ME, Rodger RA, et al. Evidence for association of an interleukin 23 receptor variant independent of the R381Q variant with rheumatoid arthritis. Ann Rheum Dis. 2009;68:1340–1344.
- Chang M, Saiki RK, Cantanese JJ, et al. The inflammatory disease-associated variants in IL12B and IL23R are not associated with rheumatoid arthritis. Arthritis Rheum. 2008;58:1877–1881.
- Farago B, Magyari L, Safrany E, et al. Functional variants of interleukin-23 receptor gene confer risk for rheumatoid arthritis but not for systemic sclerosis. Ann Rheum Dis. 2008;67:248–250.
- Orozco G, Rueda B, Robledo G, et al. Investigation of the IL23R gene in a Spanish rheumatoid arthritis cohort. Hum Immunol. 2007;68:681–684.
- Bogunia-Kubik K, Swierkot J, Malak A, et al. IL-17A, IL-17F and IL-23R gene polymorphisms in Polish patients with rheumatoid arthritis. Arch Immunol Ther Exp (Warsz). 2015;63:215–221.
- Hamdy G, Darweesh H, Khattab EA, et al. Evidence of association of interleukin-23 receptor gene polymorphisms with Egyptian rheumatoid arthritis patients. Hum Immunol. 2015;76:417–420.
- Szabo M, Safrany E, Pazar B, et al. Marked diversity of IL23R gene haplotype variants in rheumatoid arthritis comparing with Crohn's disease and ankylosing spondylitis. Mol Biol Rep. 2013;40:359–363.
- Oppmann B, Lesley R, Blom B, et al. Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12. Immunity. 2000;13:715–725.
- McInnes IB, Leung BP, Sturrock RD, et al. Interleukin-15 mediates T cell-dependent regulation of tumor necrosis factor-alpha production in rheumatoid arthritis. Nat Med. 1997;3:189–195.
- Brombacher F, Kastelein RA, Alber G. Novel IL-12 family members shed light on the orchestration of Th1 responses. Trends Immunol. 2003;24:207–212.
- Kullberg MC, Jankovic D, Feng CG, et al. IL-23 plays a key role in Helicobacter hepaticus-induced T cell-dependent colitis. J Exp Med. 2006;203:2485–2494.