Revealing of stomach histology of beta-agonist free, hygienic, and natural swine

References

• Amory, J. R., Mackenzie, A. M., & Pearce, G. P. (2006). Factors in the housing environment of finisher pigs associated with the development of gastric ulcers. The Veterinary Record, 158(8), 260–13. https://doi.org/10.1136/vr.158.8.260
   PubMed Web of Science ®Google Scholar
• Angkathunyakul, N., Treepongkaruna, S., Molagool, S., & Ruangwattanapaisarn, N. (2015). Abnormal layering of muscularis propria as a cause of chronic intestinal pseudo-obstruction: A case report and literature review. World Journal of Gastroenterology: WJG, 21(22), 7059–7064. https://doi.org/10.3748/wjg.v21.i22.7059
   PubMed Web of Science ®Google Scholar
• Bauer, M., Morales-Orcajo, E., Klemm, L., Seydewitz, R., Fiebach, V., Siebert, T., & Böl, M. (2020). Biomechanical and microstructural characterisation of the porcine stomach wall: Location- and layer-dependent investigations. Acta Biomaterialia, 102, 83–99. https://doi.org/10.1016/j.actbio.2019.11.038
   PubMed Web of Science ®Google Scholar
• Chayanisa Chaicharoen, O. H., Thorsuwan, S., Jaikua, W., Monteil, A., & Promsuban, C. (2019). Histological study of natural, beta-agonist free, and hygienic pork livers. International Journal of Applied Biomedical Engineering, 12(2), 1–7.
   Google Scholar
• De Witte, C., Ducatelle, R., & Haesebrouck, F. (2018). The role of infectious agents in the development of porcine gastric ulceration. Veterinary Journal (London, England: 1997), 236, 56–61. https://doi.org/10.1016/j.tvjl.2018.04.015
   PubMed Web of Science ®Google Scholar
• Doster, A. R. (2000). Porcine Gastric Ulcer. The Veterinary Clinics of North America Food Animal Practice, 16(1), 163–174. https://doi.org/10.1016/S0749-0720(15)30141-9
   PubMed Web of Science ®Google Scholar
• Hessing, M. J., Geudeke, M. J., Scheepens, C. J., Tielen, M. J., Schouten, W. G., & Wiepkema, P. R. (1992). Mucosal lesions in the pars esophagus in swine: Prevalence and the effect of stress. Tijdschrift voor diergeneeskunde, 117(15–16), 445–450.
   PubMed Web of Science ®Google Scholar
• Kernkamp, H. C. (1945). Gastric Ulcer in Swine. The American Journal of Pathology, 21(1), 111–113.
   PubMed Web of Science ®Google Scholar
• Knezević, M., Aleksić-Kovacević, S., & Aleksić, Z. (2007). Cell proliferation in pathogenesis of esophagogastric lesions in pigs. International Review of Cytology, 260, 1–34. https://doi.org/10.1016/s0074-7696(06)60001-6
   PubMedGoogle Scholar
• Kopinski, J. S., & McKenzie, R. A. (2007). Oesophagogastric ulceration in pigs: A visual morphological scoring guide. Australian Veterinary Journal, 85(9), 356–361. https://doi.org/10.1111/j.1751-0813.2007.196_1.x
   PubMed Web of Science ®Google Scholar
• Maeda, H., Sasaki, F., Morinaga, Y., Kabayama, M., Iwaya, H., Komaki, Y., Arima, S., Nasu, Y., Tanoue, S., Hashimoto, S., Kanmura, S., Nishiguchi, A., Taguchi, T., & Ido, A. (2021). Covering post-endoscopic submucosal dissection ulcers in miniature swine with hexanoyl (Hx: C6) group-modified alkaline-treated gelatin porous film (hag) induces proper healing by decreasing inflammation and fibrosis. Digestion, 102(3), 415–427. https://doi.org/10.1159/000509056
   PubMed Web of Science ®Google Scholar
• Martínez-Miró, S., Tecles, F., Ramón, M., Escribano, D., Hernández, F., Madrid, J., Orengo, J., Martínez-Subiela, S., Manteca, X., & Cerón, J. J. (2016). Causes, consequences and biomarkers of stress in swine: An update. BMC Veterinary Research, 12(1), 171. https://doi.org/10.1186/s12917-016-0791-8
   PubMedGoogle Scholar
• Melnichouk, S. I. (2002). Mortality associated with gastric ulceration in swine. The Canadian Veterinary Journal La Revue Veterinaire Canadienne, 43(3), 223–225.
   PubMed Web of Science ®Google Scholar
• Mpetile, Z., Young, J. M., Gabler, N. K., Dekkers, J. C., & Tuggle, C. K. (2015). Assessing peripheral blood cell profile of Yorkshire pigs divergently selected for residual feed intake. Journal of Animal Science, 93(3), 892–899. https://doi.org/10.2527/jas.2014-8132
   PubMed Web of Science ®Google Scholar
• Omotosho, O. O., Emikpe, B. O., Lasisi, O. T., & Jarikre, T. A. (2016). Prevalence, distribution and pattern of gastric lesions in slaughtered pigs in south-western Nigeria. The Onderstepoort Journal of Veterinary Research, 83(1), a1063. https://doi.org/10.4102/ojvr.v83i1.1063
   PubMed Web of Science ®Google Scholar
• Penington, D. G. (1967). Regulation of red cell and platelet production. Proceedings of the Royal Society of Medicine, 60(10), 1032–1036. https://doi.org/10.1177/003591576706001043
   PubMedGoogle Scholar
• Peralvo-Vidal, J. M., Weber, N. R., Nielsen, J. P., Bache, J. K., Haugegaard, S., & Pedersen, A. (2021). Risk factors for gastric ulceration in nursery pigs. Preventive Veterinary Medicine, 189, 105298. https://doi.org/10.1016/j.prevetmed.2021.105298
   PubMed Web of Science ®Google Scholar
• Queiroz, D. M., Rocha, G. A., Mendes, E. N., De Moura, S. B., De Oliveira, A. M., & Miranda, D. (1996). Association between Helicobacter and gastric ulcer disease of the pars esophagea in swine. Gastroenterology, 111(1), 19–27. https://doi.org/10.1053/gast.1996.v111.pm8698198
   PubMed Web of Science ®Google Scholar
• Reese, N. A., Muggenburg, B. A., Kowalczyk, T., Grummer, R. H., & Hoekstra, W. G. (1966). Nutritional and environmental factors influencing gastric ulcers in swine. Journal of Animal Science, 25(1), 14–20. https://doi.org/10.2527/jas1966.25114x
   PubMed Web of Science ®Google Scholar
• Reiner, G., Fischer, R., Hepp, S., Berge, T., Köhler, F., & Willems, H. (2007). Quantitative trait loci for red blood cell traits in swine. Animal Genetics, 38(5), 447–452. https://doi.org/10.1111/j.1365-2052.2007.01629.x
   PubMed Web of Science ®Google Scholar
• Roels, S., Ducatelle, R., & Broekaert, D. (1997). Keratin pattern in hyperkeratotic and ulcerated gastric pars oesophagea in pigs. Research in Veterinary Science, 62(2), 165–169. https://doi.org/10.1016/s0034-5288(97)90140-5
   PubMed Web of Science ®Google Scholar
• Sikiric, P., & Brzozowski, T. (2020). Physiological and pharmacological mechanisms in gastrointestinal protection, ulcer healing and mucosal repair - an update. Current Pharmaceutical Design, 26(25), 2933–2935. https://doi.org/10.2174/138161282625200629111007
   PubMed Web of Science ®Google Scholar
• Stygar, A. H., Chantziaras, I., Toppari, I., Maes, D., & Niemi, J. K. (2020). High biosecurity and welfare standards in fattening pig farms are associated with reduced antimicrobial use. Animal, 14(10), 2178–2186. https://doi.org/10.1017/s1751731120000828
   PubMed Web of Science ®Google Scholar
• van Erp, R. J. J., van Hees, H. M. J., Zijlstra, R. T., van Kempen, T., van Klinken, J. B., & Gerrits, W. J. J. (2018). Reduced feed intake, rather than increased energy losses, explains variation in growth rates of normal-birth-weight piglets. The Journal of Nutrition, 148(11), 1794–1803. https://doi.org/10.1093/jn/nxy200
   PubMed Web of Science ®Google Scholar
• Yun, J., Muurinen, J., Nykäsenoja, S., Seppä-Lassila, L., Sali, V., Suomi, J., Tuominen, P., Joutsen, S., Hämäläinen, M., Olkkola, S., Myllyniemi, A. L., Peltoniemi, O., & Heinonen, M. (2021). Antimicrobial use, biosecurity, herd characteristics, and antimicrobial resistance in indicator Escherichia coli in ten Finnish pig farms. Preventive Veterinary Medicine, 193, 105408. https://doi.org/10.1016/j.prevetmed.2021.105408
   PubMed Web of Science ®Google Scholar