References
- Rivera GK, Crist WM. Acute lymphoblastic leukemia. In: , Handin R I, Stossel T P, Lux S E, ed, editors. Blood. Principles and practice of hematology. Philadelphia, PA: J. B. Lippincott Co.; 1995. p.743–59.
- Uckun FM, Sensel MG, Sun L, Steinherz PG, Trigg ME, Heerema NA. Biology and treatment of childhood T-lineage acute lymphoblastic leukemia. Blood 1998;91:735–46.
- Morris JC, Waldmann TA, Janik JE. Receptor-directed therapy of T-cell leukemias and lymphomas. J Immunotoxicol 2008;5:235–48.
- Dearden CE, Matutes E, Cazin B, Tjønnfjord GE, Parreira A, Nomdedeu B, et al.. High remission rate in T-cell prolymphocytic leukemia with CAMPATH-1H. Blood 2001;98:1721–6.
- Rodig SJ, Abramson JS, Pinkus GS, Treon SP, Dorfman DM, Dong HY, et al.. Heterogeneous CD52 expression among hematologic neoplasms: implications for the use of alemtuzumab (CAMPATH-1H). Clin Cancer Res 2006;12:7174–9.
- Ravandi F, O’Brien S. Alemtuzumab in CLL and other lymphoid neoplasms. Cancer Invest 2006, 24:718–25.
- Ishida T, Ueda R. CCR4 as a novel molecular target for immunotherapy of cancer. Cancer Sci 2006;97:1139–46.
- Peipp M, Küpers H, Saul D, Schlierf B, Greil J, Zunino SJ, et al.. A recombinant CD7-specific single-chain immunotoxin is a potent inducer of apoptosis in acute leukemic T cells. Cancer Res 2002;62:2848–55.
- Sato T, Yamochi T. CD26 regulates p38 mitogen-activated protein kinase-dependent phosphorylation of integrin beta, adhesion to extracellular matrix, and tumorigenicity of T-anaplastic large cell lymphoma Karpas 299. Cancer Res 2005;65:6950–6.
- Steinbach D, Wittig S, Cario G, Viehmann S, Mueller A, Gruhn B, et al.. The multidrug resistance-associated protein 3 (MRP3) is associated with a poor outcome in childhood ALL and may account for the worse prognosis in male patients and T-cell immunophenotype. Blood 2003;102:4493–8.
- McKallip RJ, Lombard C, Fisher M, Martin BR, Ryu S, Grant S, et al.. Targeting CB2 cannabinoid receptors as a novel therapy to treat malignant lymphoblastic disease. Blood 2002;100:627–34.
- Villalba M, Altman A. Protein kinase C-theta (PKCtheta), a potential drug target for therapeutic intervention with human T cell leukemias. Curr Cancer Drug Targets 2002;2:125–37.
- Elbashir S, Harborth J, Lendeckel W, Yalcin A, Weber K, Tuschl T. Duplexes of 21-nucleotide RNAs mediate RNA interference in cultured mammalian cells. Nature 2001;411:494–8.
- Oh YK, Park TG. siRNA delivery systems for cancer treatment. Adv Drug Deliv Rev 2009;61:850–62.
- Devi RS. siRNA-based approaches in cancer therapy. Cancer Gene Ther 2006;13:819–29.
- Whitehead KA, Langer R, Anderson DG. Knocking down barriers: advances in siRNA delivery. Nat Rev Drug Discov 2009;8:129–38.
- Cioca DP, Aoki Y, Kijosawa K. RNA interference is a functional pathway with therapeutic potential in human myeloid leukemia cell lines. Cancer Gene Ther 2003;10:125–33.
- Li L, Leid M, Rothenberg EV. An early T cell lineage commitment checkpoint dependent on the transcription factor Bcl11b. Science 2010;329:89–93.
- Wakabayashi Y, Watanabe H, Inoue J, Takeda N, Sakata J, Takeda N, et al.. Bcl11bis required for differentiation and survival of αβ T lymphocytes. Nat Immunol 2003;4:533–9.
- Cismasiu VB, Ghanta S, Duque J, Albu D, Chen HM, Kasturi R, et al.. BCL11Bparticipates in the activation of interleukin-2 gene expression in CD4+ T lymphocytes. Blood 2006;108:2695–702.
- Liu P, Li P, Burke S. Critical roles of Bcl11b in T-cell development and maintenance of T-cell identity. Immunol Rev 2010;238:138–49.
- Przybylski GK, Dik WA, Wanzeck J, Grabarczyk P, Majunke S, Martin-Subero JI, et al.. Disruption of the BCL11B gene through inv(14)(q11·2q32·31) results in the expression of BCL11B-TRDC fusion transcripts and is associated with the absence of wild-type BCL11B transcripts in T-ALL. Leukemia 2005;19:201–8.
- Karlsson A, Nordigården A, Jönsson JI, Söderkvist P.Bcl11bmutations identified in murine lymphomas increase the proliferation rate of hematopoietic progenitor cells. BMC Cancer 2007;7:195.
- Su XY, Della-Valle V, Andre-Schmutz I, Lemercier C, Radford-Weiss I, Ballerini P, et al.. HOX11L2/TLX3 is transcriptionally activated through T-cell regulatory elements downstream of BCL11B as a result of the t(5;14)(q35;q32). Blood 2006;108:4198–201.
- Oshiro A, Tagawa H, Ohshima K, Karube K, Uike N, Tashiro Y, et al.. Identification of subtype-specific genomic alterations in aggressive adult T-cell leukemia/lymphoma. Blood 2006;107:4500–7.
- Huang X, Chen S, Shen Q, Yang LJ, Li B, Zhong LY, et al.. Analysis of the expression pattern of the BCL11B gene and its relatives in patients with T-cell acute lymphoblastic leukemia. J Hematol Oncol 2010;3:44.
- Grabarczyk P, Przybylski GK, Depke M, Völker U, Bahr J, Assmus K, et al.. Inhibition of BCL11B expression leads to apoptosis of malignant but not normal mature T cells. Oncogene 2007;26:3797–810.
- Heidenreich O. Oncogene suppression by small interfering RNAs. Curr Pharm Biotechnol 2004;5:349–54.
- Avram D, Fields A, Pretty On Top K, Nevrivy DJ, Ishmael JE, Leid M. Isolation of a novel family of C2H2 zinc finger proteins implicated in transcriptional repression mediated by chicken ovalbumin upstream promoter transcription factor (COUP-TF) orphan nuclear receptors. J Biol Chem 2000;275:10315–22.
- Onciu M, Lai R, Vega F, Bueso-Ramos C, Medeiros LJ. Precursor T-cell acute lymphoblastic leukemia in adults: age-related immunophenotypic, cytogenetic, and molecular subsets. Am J Clin Pathol 2002;117:252–8.