References
- Haffner SM, Lehto S, Ronnemaa T, Pyorala K, Laakso M. Mortality from coronary heart disease in subjects with Type 2 diabetes and in nondiabetic subjects with and without prior myocardial infarction. N. Engl. J. Med.339, 229–234 (1998).
- Hayat SA, Patel B, Khattar RS, Malik RA. Diabetic cardiomyopathy: mechanisms, diagnosis and treatment. Clin. Sci.107, 539–557 (2004).
- Aneja A, Tang WH, Bansilal S, Garcia MJ, Farkouh ME. Diabetic cardiomyopathy: insights into pathogenesis, diagnostic challenges, and therapeutic options. Am. J. Med.121, 748–757 (2004).
- Lieb W, Xanthakis V, Sullivan LM et al. Longitudinal tracking of left ventricular mass over the adult life course: clinical correlates of short- and long-term change in the framingham offspring study. Circulation119, 3085–3092 (2009).
- Borisov AB, Ushakov AV, Zagorulko AK et al. Intracardiac lipid accumulation, lipoatrophy of muscle cells and expansion of myocardial infarction in Type 2 diabetic patients. Micron39, 944–951 (2008).
- van Heerebeek L, Hamdani N, Handoko ML et al. Diastolic stiffness of the failing diabetic heart: importance of fibrosis, advanced glycation end products, and myocyte resting tension. Circulation117, 43–51 (2008).
- Gertz EW, Wisneski JA, Stanley WC, Neese RA. Myocardial substrate utilization during exercise in humans. Dual carbon-labeled carbohydrate isotope experiments. J. Clin. Invest.82, 2017–25 (1988).
- von Lewinski D, Gasser R, Rainer PP et al. Functional effects of glucose transporters in human ventricular myocardium. Eur. J. Heart Fail.12, 106–113 (2010).
- Murray AJ, Anderson RE, Watson GC, Radda GK, Clarke K. Uncoupling proteins in human heart. Lancet364, 1786–1788 (2004).
- Peterson LR, Herrero P, Schechtman KB et al. Effect of obesity and insulin resistance on myocardial substrate metabolism and efficiency in young women. Circulation109, 2191–2196 (2004).
- Monnier L, Mas E, Ginet C et al. Activation of oxidative stress by acute glucose fluctuations compared with sustained chronic hyperglycemia in patients with Type 2 diabetes. JAMA295, 1681–1687 (2006).
- Brownlee M. Biochemistry and molecular cell biology of diabetic complications. Nature414, 813–820 (2001).
- Hotamisligil GS. Inflammation and metabolic disorders. Nature444, 860–867 (2006).
- Houstis N, Rosen ED, Lander ES. Reactive oxygen species have a causal role in multiple forms of insulin resistance. Nature440, 944–948 (2006).
- Kim JA, Montagnani M, Koh KK, Quon MJ. Reciprocal relationships between insulin resistance and endothelial dysfunction: molecular and pathophysiological mechanisms. Circulation113, 1888–1904 (2006).
- Rask-Madsen C, Dominguez H, Ihlemann N, Hermann T, Kober L, Torp-Pedersen C. Tumor necrosis factor-α inhibits insulin’s stimulating effect on glucose uptake and endothelium-dependent vasodilation in humans. Circulation108, 1815–1821 (2003).
- Kim F, Gallis B, Corson MA. TNF-α inhibits flow and insulin signaling leading to NO production in aortic endothelial cells. Am. J. Physiol. Cell Physiol.280, C1057–C1065 (2001).
- Picchi A, Gao X, Belmadani S et al. Tumor necrosis factor-α induces endothelial dysfunction in the prediabetic metabolic syndrome. Circ. Res.99, 69–77 (2006).