References
- Naldi L, Rebora A. Clinical practice. Seborrheic dermatitis. N Engl J Med. 2009;360:387–396. doi:10.1056/NEJMcp080646419164189
- Sampaio ALSB, Mameri ACA, Vargas TJDS, et al. Seborrheic dermatitis. An Bras Dermatol. 2011;86:1061–71; quiz 1072–4. doi:10.1590/s0365-05962011000600002
- Dessinioti C, Katsambas A. Seborrheic dermatitis: etiology, risk factors, and treatments: facts and controversies. Clin Dermatol. 2013;31:343–351. doi:10.1016/j.clindermatol.2013.01.00123806151
- Prete SD, Angeli A, Ghobril C, et al. Anion inhibition profile of the β-carbonic anhydrase from the opportunist pathogenic fungus malassezia restricta involved in dandruff and seborrheic dermatitis. Metabolites. 2019;9:147. doi:10.3390/metabo9070147
- Wikramanayake TC, Borda LJ, Miteva M, et al. Seborrheic dermatitis-looking beyond Malassezia. Exp Dermatol. 2019;28:991–1001. doi:10.1111/exd.1400631310695
- Forrestel AK, Kovarik CL, Mosam A, et al. Diffuse HIV-associated seborrheic dermatitis – a case series. Int J STD AIDS. 2016;27:1342–1345. doi:10.1177/095646241664181627013615
- Passi S, De Luca C, Picardo M, et al. Deficit ematico di acidi grassi polinsaturi di fosfolipidi, vitamina E e glutatione perossidasi come possibile fattore di rischio nell'insorgenza e nello sviluppo della sindrome da immunodeficienza acquisita. [Blood deficiency values of polyunsaturated fatty acids of phospholipids, vitamin E and glutathione peroxidase as possible risk factors in the onset and development of acquired immunodeficiency syndrome]. G Ital Dermatol Venereol. 1990;125:125–130. Italian.2228037
- Emre S, Metin A, Demirseren DD, et al. The association of oxidative stress and disease activity in seborrheic dermatitis. Arch Dermatol Res. 2012;304:683–687. doi:10.1007/s00403-012-1254-022699428
- Filomeni G, Rotilio G, Ciriolo MR. Cell signalling and the glutathione redox system. Biochem Pharmacol. 2002;64:1057–1064. doi:10.1016/s0006-2952(02)01176-012213605
- Forman HJ, Zhang H, Rinna A. Glutathione: overview of its protective roles, measurement, and biosynthesis. Mol Aspects Med. 2009;30:1–12. doi:10.1016/j.mam.2008.08.00618796312
- Leedle RA, Aust SD. The effect of glutathione on the vitamin E requirement for inhibition of liver microsomal lipid peroxidation. Lipids. 1990;25:241–245. doi:10.1007/bf025443822352452
- Limongi D, Baldelli S, Checconi P, et al. GSH-C4 acts as anti-inflammatory drug in different models of canonical and cell autonomous inflammation through NFκB inhibition. Front Immunol. 2019;10:155. doi:10.3389/fimmu.2019.0015530787932
- Poór AK, Brodszky V, Péntek M, et al. Is the DLQI appropriate for medical decision-making in psoriasis patients? Arch Dermatol Res. 2018;310:47–55. doi:10.1007/s00403-017-1794-429128966
- Palamara AT, Brandi G, Rossi L, et al. New synthetic glutathione derivatives with increased antiviral activities. Antivir Chem Chemother. 2004;15:83–91. doi:10.1177/09563202040150020415185726
- Ninfali P, Antonini E, Frati A, et al. C-Glycosyl Flavonoids from Beta vulgaris Cicla and Betalains from Beta vulgaris rubra: antioxidant, anticancer and antiinflammatory activities-A review. Phytother Res. 2017;31:871–884. doi:10.1002/ptr.581928464411
- Clark GW, Pope SM, Jaboori KA. Diagnosis and treatment of seborrheic dermatitis. Am Fam Physician. 2015;91:185–190.25822272
- Schlesinger T, Powell CR. Efficacy and safety of a low molecular weight hyaluronic acid topical gel in the treatment of facial seborrheic dermatitis final report. J Clin Aesthet Dermatol. 2014;7:15–18.24847403
- Gariboldi S, Palazzo M, Zanobbio L, et al. Low molecular weight hyaluronic acid increases the self-defense of skin epithelium by induction of beta-defensin 2 via TLR2 and TLR4. J Immunol. 2008;181:2103–2110. doi:10.4049/jimmunol.181.3.210318641349