Association of Mannose-Binding Lectin 2 Gene Polymorphism with Tuberculosis Based on Mycobacterium tuberculosis Lineages

References

• WorId Health Organization. Global Tuberculosis Report 2020. Geneva: World Health Organization; 2020.
   Google Scholar
• Yong Tan S, Kwok E. Albert Calmette (1863–1933): originator of the BCG vaccine. Singapore Med J. 2012;53:433–434.
   Web of Science ®Google Scholar
• Towey F. Historical Profile Leon Charles Albert Calmette and Jean-Marie Camille Guerin. Lancet Respir Med. 2015;3:186–187. doi:10.1016/S2213-2600(15)00065-X
   Web of Science ®Google Scholar
• Sakula A. BCG: who were Calmette and Guérin?. Thorax. 1983;38:806–812.
   PubMed Web of Science ®Google Scholar
• Zhang JX, Gong WP, Zhu DL, et al. Mannose-binding lectin 2 gene polymorphisms and their association with tuberculosis in a Chinese population. Infect Dis Poverty. 2020;9:46. doi:10.1186/s40249-020-00664-9
   Web of Science ®Google Scholar
• Ceylan E, Karkucak M, Coban H, et al. Evaluation of TNF-alpha gene (G308A) and MBL2 gene codon 54 polymorphisms in Turkish patients with tuberculosis. J Infect Public Health. 2017;10:774–777. doi:10.1016/j.jiph.2016.11.003
   PubMed Web of Science ®Google Scholar
• Hu CY. X-Linked Susceptible Genes and Genome-Wide Association Study of Pulmonary Tuberculosis. Beijing: Chinese People’s Liberation Army Academy of Military Sciences; 2015.
   Google Scholar
• Dos Santos Silva PM, de Oliveira WF, Albuquerque PBS, et al. Insights into anti-pathogenic activities of mannose lectins. Int J Biol Macromol. 2019;140:234–244.
   Web of Science ®Google Scholar
• Kilpatrick DC. Animal lectins: a historical introduction and overview. Biochim Biophys Acta. 2002;1572:187–197. doi:10.1016/S0304-4165(02)00308-2
   PubMed Web of Science ®Google Scholar
• Holmskov U, Thiel S, Jensenius JC. Collections and ficolins: humoral lectins of the innate immune defense. Annu Rev Immunol. 2003;21:547–578. doi:10.1146/annurev.immunol.21.120601.140954
   PubMed Web of Science ®Google Scholar
• Garred P, Larsen F, Seyfarth J, et al. Mannose-binding lectin and its genetic variants. Genes Immun. 2006;7(2):85–94.
   Web of Science ®Google Scholar
• Heitzeneder S, Seidel M, Förster-Waldl E, et al. Mannan-binding lectin deficiency - good news, bad news, doesn’t matter?. Clin Immunol. 2012;143(1):22–38. doi:10.1016/j.clim.2011.11.002
   PubMed Web of Science ®Google Scholar
• Zheng M, Shi S, Wei W, et al. Correlation between MBL2/CD14/TNF-α gene poly-morphisms and susceptibility to spinal tuberculosis in Chinese population. Biosci Rep. 2018;38:BSR20171140. doi:10.1042/BSR20171140
   Web of Science ®Google Scholar
• Amiri A, Sabooteh T, Shahsavar F, et al. Mannose-Binding Lectin (MBL) gene polymorphisms in susceptibility to pulmonary tuberculosis among the lur population of Lorestan Province of Iran. Genom Data. 2017;12:146–150.
   PubMedGoogle Scholar
• Liu Q, Wei J, Li Y, et al. Mycobacterium tuberculosis clinical isolates carry mutational signatures of host immune environments. Sci Adv. 2020;6:eaba4901. doi:10.1126/sciadv.aba4901
   PubMed Web of Science ®Google Scholar
• Omae Y, Toyo-Oka L, Yanai H, et al. Pathogen lineage-based genome-wide association study identified CD53 as susceptible locus in tuberculosis. J Hum Genet. 2017;62:1015–1022. doi:10.1038/jhg.2017.82
   Web of Science ®Google Scholar
• National Health and Family Planning Commission of the People’s Republic of China, WS 288–2017. Diagnosis for Pulmonary Tuberculosis. Beijing: People’s Medical Publishing House; 2017.
   Google Scholar
• Honore S, Vincensini JP, Hocqueloux L, et al. Diagnostic value of a nested polymerase chain reaction assay on peripheral blood mononuclear cells from patients with pulmonary and extrapulmonary tuberculosis. Int J Tuberc Lung Dis. 2001;5:754–762.
   PubMed Web of Science ®Google Scholar
• Letunic I, Bork P. Interactive tree of life (iTOL) v5: an online tool for phylogenetic tree display and annotation. Nucleic Acids Res. 2021;49(W1):W293–W296. doi:10.1093/nar/gkab301
   PubMed Web of Science ®Google Scholar
• Thakkinstian A, McElduff P, D’Este C, et al. A method for meta-analysis of molecular association studies. Stat Med. 2005;24:1291–1306. doi:10.1002/sim.2010
   PubMed Web of Science ®Google Scholar
• Woolhouse ME, Webster JP, Domingo E, et al. Biological and biomedical implications of the co-evolution of pathogens and their hosts. Nat Genet. 2002;32:569–577. doi:10.1038/ng1202-569
   PubMed Web of Science ®Google Scholar
• Ebert D, Fields PD. Host-parasite co-evolution and its genomic signature. Nat Rev Genet. 2020;21:754–768.
   Web of Science ®Google Scholar
• Ahlawat N, Geeta Arun M, Maggu K, et al. Enemies make you stronger: co-evolution between fruit fly host and bacterial pathogen increases postinfection survivorship in the host. Ecol Evol. 2021;11:9563–9574. doi:10.1002/ece3.7774
   Web of Science ®Google Scholar
• Ebert D. Open questions: what are the genes underlying antagonistic coevolution?. BMC Biol. 2018;16:114. doi:10.1186/s12915-018-0583-7
   Web of Science ®Google Scholar
• Nadesalingam J, Dodds AW, Reid KB, et al. Mannose-binding lectin recognizes peptidoglycan via the N-acetyl glucosamine moiety, and inhibits ligand-induced proinflammatory effect and promotes chemokine production by macrophages. J Immunol. 2005;175:1785–1794. doi:10.4049/jimmunol.175.3.1785
   Web of Science ®Google Scholar
• Michelle A. Mannose-binding lectin levels and susceptibility to tuberculosis. Clin Immunol. 2010;135:S79–S79. doi:10.1016/j.clim.2010.03.238
   Web of Science ®Google Scholar
• Søborg C, Madsen HO, Andersen AB, et al. Mannose-binding lectin polymorphisms in clinical tuberculosis. J Infect Dis. 2003;188:777–782. doi:10.1086/377183
   PubMed Web of Science ®Google Scholar
• Guo YL, Liu Y, Ban WJ, et al. Association of mannose-binding lectin gene polymorphisms with the development of pulmonary tuberculosis in China. BMC Infect Dis. 2017;17:210. doi:10.1186/s12879-017-2310-3
   Web of Science ®Google Scholar
• Chen M, Liang Y, Li W, et al. Impact of MBL and MASP-2 gene polymorphism and its interaction on susceptibility to tuberculosis. BMC Infect Dis. 2015;15:151. doi:10.1186/s12879-015-0879-y
   PubMed Web of Science ®Google Scholar
• Chen H, He L, Cai C, et al. Characteristics of distribution of Mycobacterium tuberculosis lineages in China. Sci China Life Sci. 2018;61:651–659. doi:10.1007/s11427-017-9243-0
   Web of Science ®Google Scholar
• Glynn JR, Whiteley J, Bifani PJ, et al. Worldwide occurrence of Beijing/W strains of Mycobacterium tuberculosis: a systematic review. Emerg Infect Dis. 2002;8(8):843–849. doi:10.3201/eid0808.020002
   PubMed Web of Science ®Google Scholar
• Karmakar M, Trauer JM, Ascher DB, Denholm JT. Hyper transmission of Beijing lineage Mycobacterium tuberculosis: systematic review and meta-analysis. J Infect. 2019;79(6):572–581.
   Web of Science ®Google Scholar
• Wang C, Peyron P, Mestre O, et al. Innate immune response to Mycobacterium tuberculosis Beijing and other genotypes. PLoS One. 2010;5:e13594. doi:10.1371/journal.pone.0013594
   PubMed Web of Science ®Google Scholar
• Chen YY, Chang JR, Huang WF, et al. The pattern of cytokine production in vitro induced by ancient and modern Beijing Mycobacterium tuberculosis strains. PLoS One. 2014;9:e94296. doi:10.1371/journal.pone.0094296
   PubMed Web of Science ®Google Scholar
• Müller SJ, Schurz H, Tromp G, et al. A multi-phenotype genome-wide association study of clades causing tuberculosis in a Ghanaian- and South African cohort. Genomics. 2021;113:1802–1815. doi:10.1016/j.ygeno.2021.04.024
   Web of Science ®Google Scholar