Identification of Oxidative Stress-Related Biomarkers in Acute Myocardial Infarction

References

• Liu B, Chen Y, St Clair DK. ROS and p53: a versatile partnership. Free Radic Biol Med. 2008;44(8):1529–1535. doi:10.1016/j.freeradbiomed.2008.01.011
   PubMed Web of Science ®Google Scholar
• Suematsu N, Tsutsui H, Wen J, et al. Oxidative stress mediates tumor necrosis factor-alpha-induced mitochondrial DNA damage and dysfunction in cardiac myocytes. Circulation. 2003;107(10):1418–1423. doi:10.1161/01.CIR.0000055318.09997.1F
   PubMed Web of Science ®Google Scholar
• Cailleret M, Amadou A, Andrieu-Abadie N, et al. N-acetylcysteine prevents the deleterious effect of tumor necrosis factor-(alpha) on calcium transients and contraction in adult rat cardiomyocytes. Circulation. 2004;109(3):406–411. doi:10.1161/01.CIR.0000109499.00587.FF
   PubMed Web of Science ®Google Scholar
• Chen L, Gong P, Su Y, et al. Angiotensin II type 2 receptor agonist attenuates LPS-induced acute lung injury through modulating THP-1-derived macrophage reprogramming. Naunyn Schmiedebergs Arch Pharmacol. 2023. doi:10.1007/s00210-023-02589-0
   Web of Science ®Google Scholar
• Lagan J, Naish JH, Fortune C, et al. Myocardial involvement in eosinophilic granulomatosis with polyangiitis evaluated with cardiopulmonary magnetic resonance. Int J Cardiovasc Imaging. 2021;37(4):1371–1381. doi:10.1007/s10554-020-02091-1
   PubMed Web of Science ®Google Scholar
• Cheng Y, Luo D, Zhao Y, Rong J. N-Propargyl caffeate amide (PACA) prevents cardiac fibrosis in experimental myocardial infarction by promoting pro-resolving macrophage polarization. Aging. 2020;12(6):5384–5398. doi:10.18632/aging.102959
   PubMedGoogle Scholar
• Liu LL, Lu JL, Chao PL, Lin LR, Zhang ZY, Yang TC. Lower prevalence of circulating invariant natural killer T (iNKT) cells in patients with acute myocardial infarction undergoing primary coronary stenting. Int Immunopharmacol. 2011;11(4):480–484. doi:10.1016/j.intimp.2010.12.019
   PubMed Web of Science ®Google Scholar
• Smekal A, Vaclavik J. Adipokines and cardiovascular disease: a comprehensive review. Biomed Pap Med Fac Univ Palacky Olomouc Czech Repub. 2017;161(1):31–40. doi:10.5507/bp.2017.002
   PubMed Web of Science ®Google Scholar
• Zhou F, Liu D, Ning HF, Yu XC, Guan XR. The roles of p62/SQSTM1 on regulation of matrix metalloproteinase-9 gene expression in response to oxLDL in atherosclerosis. Biochem Biophys Res Commun. 2016;472(3):451–458. doi:10.1016/j.bbrc.2016.01.065
   PubMed Web of Science ®Google Scholar
• Garvin P, Jonasson L, Nilsson L, Falk M, Kristenson M, Li Y. Plasma matrix metalloproteinase-9 levels predict first-time coronary heart disease: an 8-year follow-up of a community-based middle aged population. PLoS One. 2015;10(9):e0138290. doi:10.1371/journal.pone.0138290
   PubMed Web of Science ®Google Scholar
• Tan X, Dai Q, Sun H, et al. Systematic bioinformatics analysis based on public and second-generation sequencing transcriptome data: a study on the diagnostic value and potential mechanisms of immune-related genes in acute myocardial infarction. Front Cardiovasc Med. 2022;9:863248. doi:10.3389/fcvm.2022.863248
   PubMed Web of Science ®Google Scholar
• Xie J, Luo C, Mo B, et al. Inflammation and oxidative stress role of S100A12 as a potential diagnostic and therapeutic biomarker in acute myocardial infarction. Oxid Med Cell Longev. 2022;2022:2633123. doi:10.1155/2022/2633123
   PubMed Web of Science ®Google Scholar
• Wallace DC, Fan W, Procaccio V. Mitochondrial energetics and therapeutics. Annu Rev Pathol. 2010;5(1):297–348. doi:10.1146/annurev.pathol.4.110807.092314
   PubMed Web of Science ®Google Scholar
• Kumar V, Prabhu SD, Bansal SS. CD4(+) T-lymphocytes exhibit biphasic kinetics post-myocardial infarction. Front Cardiovasc Med. 2022;9:992653. doi:10.3389/fcvm.2022.992653
   PubMedGoogle Scholar
• Lillo R, Graziani F, Franceschi F, et al. Inflammation across the spectrum of hypertrophic cardiac phenotypes. Heart Fail Rev. 2023;28(5):1065–1075. doi:10.1007/s10741-023-10307-4
   PubMed Web of Science ®Google Scholar
• Becker RC, Owens AP, Sadayappan S. Tissue-level inflammation and ventricular remodeling in hypertrophic cardiomyopathy. J Thromb Thrombolysis. 2020;49(2):177–183. doi:10.1007/s11239-019-02026-1
   PubMed Web of Science ®Google Scholar
• Matthia EL, Setteducato ML, Elzeneini M, et al. Circulating biomarkers in hypertrophic cardiomyopathy. J Am Heart Assoc. 2022;11(23):e027618. doi:10.1161/JAHA.122.027618
   PubMed Web of Science ®Google Scholar
• Kumar V, Rosenzweig R, Asalla S, Nehra S, Prabhu SD, Bansal SS. TNFR1 contributes to activation-induced cell death of pathological CD4(+) T lymphocytes during ischemic heart failure. JACC Basic Transl Sci. 2022;7(10):1038–1049. doi:10.1016/j.jacbts.2022.05.005
   PubMedGoogle Scholar
• Peet C, Ivetic A, Bromage DI, Shah AM. Cardiac monocytes and macrophages after myocardial infarction. Cardiovasc Res. 2020;116(6):1101–1112. doi:10.1093/cvr/cvz336
   PubMed Web of Science ®Google Scholar
• Prager I, Watzl C. Mechanisms of natural killer cell-mediated cellular cytotoxicity. J Leukoc Biol. 2019;105(6):1319–1329. doi:10.1002/JLB.MR0718-269R
   PubMed Web of Science ®Google Scholar
• Knorr M, Munzel T, Wenzel P. Interplay of NK cells and monocytes in vascular inflammation and myocardial infarction. Front Physiol. 2014;5:295. doi:10.3389/fphys.2014.00295
   PubMed Web of Science ®Google Scholar
• Fiorelli S, Anesi A, Porro B, et al. Lipidomics analysis of monocytes from patients with acute myocardial infarction reveals lactosylceramide as a new player in monocyte migration. FASEB J. 2021;35(5):e21494. doi:10.1096/fj.202001872RRR
   PubMed Web of Science ®Google Scholar
• Swirski FK, Pittet MJ, Kircher MF, et al. Monocyte accumulation in mouse atherogenesis is progressive and proportional to extent of disease. Proc Natl Acad Sci U S A. 2006;103(27):10340–10345. doi:10.1073/pnas.0604260103
   PubMed Web of Science ®Google Scholar
• Potteaux S, Gautier EL, Hutchison SB, et al. Suppressed monocyte recruitment drives macrophage removal from atherosclerotic plaques of Apoe-/- mice during disease regression. J Clin Invest. 2011;121(5):2025–2036. doi:10.1172/JCI43802
   PubMed Web of Science ®Google Scholar