References
- Bhadauria, S., Singh, G., Sinha, N., & Srivastava, S. (2007). Isoniazid induces oxidative stress, mitochondrial dysfunction and apoptosis in HepG2 cells. Cell Mol Biol (Noisy-le-grand), 53, 102–114.
- Chowdhury, A., Santra, A., Bhattacharjee, K., Ghatak, S., Saha, DR., Dhali, GK. (2006). Mitochondrial oxidative stress and permeability transition in isoniazid and rifampicin induced liver injury in mice. J Hepatol, 45, 117–126.
- Finsterer, J., & Segall, L. (2010). Drugs interfering with mitochondrial disorders. Drug Chem Toxicol, 33, 138–151.
- Garone, C., Tadesse, S., & Hirano, M. (2011). Clinical and genetic spectrum of mitochondrial neurogastrointestinal encephalomyopathy. Brain, 134, 3326–3332.
- Halter, J., Schüpbach, W. M., Casali, C., Elhasid R., Fay K., Hammans S.., et al. (2011). Allogeneic hematopoietic SCT as treatment option for patients with mitochondrial neurogastrointestinal encephalomyopathy (MNGIE): A consensus conference proposal for a standardized approach. Bone Marrow Transplant, 46, 330–337.
- Ikeda, A., Ikeda, T., Ikeda, N., Kawakami, Y., Mimura, O. (2006). Leber's hereditary optic neuropathy precipitated by ethambutol. Jpn J Ophthalmol, 50, 280–283.
- Schoffner, J. (2010). Mitochondrial neurogastrointestinal encephalopathy disease. 2005 Apr 22 [Updated 2010 May 11]. In: Pagon, R.A., Bird, T.D., Dolan, C.R., et al., editors. GeneReviews™ [Internet]. Seattle (WA): University of Washington, Seattle; 1993-. Available from: http://www.ncbi.nlm.nih.gov/books/NBK1179/.
- Seo, J. H., Hwang, J. M., & Park S. S. (2010). Antituberculosis medication as a possible epigenetic factor of Leber's hereditary optic neuropathy. Clin Exp Ophthalmol, 38, 363–366.