References
- Shouman M, Abdallah N, El Tablawy N, Rashed L. Biochemical markers of endothelial dysfunction in pediatric nephritic syndrome. Arch Med Sci. 2009;5:415–421
- Domański L, Gryczman M, Pawlik A, et al. Circulating adhesion molecules during kidney allograft reperfusion. Transpl Immunol. 2006;16(3–4):172–175
- Clarkson MR, Sayegh MH. T-cell costimulatory pathways in allograft rejection and tolerance. Transplantation. 2005;80:555–563
- Kosmaczewska A, Magott-Procelewska M, Frydecka I, et al. CD40L, CD28, and CTLA-4 expression on CD4+ T cells in kidney graft recipients: a relationship with post-transplantation clinical course. Transpl Immunol. 2006;16:32–40
- Salvalaggio PR, Camirand G, Ariyan CE, et al. Antigen exposure during enhanced CTLA-4 expression promotes allograft tolerance in vivo. J Immunol. 2006;176:2292–2298
- Gregersen PK, Lee HS, Batliwalla F, et al. PTPN22: setting thresholds for autoimmunity. Semin Immunol. 2006;18:214–223
- Begovich AB, Carlton VE, Honigberg LA, et al. A missense single-nucleotide polymorphism in a gene encoding a protein tyrosine phosphatase (PTPN22) is associated with rheumatoid arthritis. Am J Hum Genet. 2004;75:330–337
- Wiśniewski A, Kusztal M, Magott-Procelewska M, et al. Possible association of cytotoxic T-lymphocyte antigen 4 gene promoter single nucleotide polymorphism with acute rejection of allogenic kidney transplant. Transplant Proc. 2006;38:56–58
- Domański L, Bobrek-Lesiakowska K, Kłoda K, et al. Lack of association of the rs2476601 PTPN22 gene polymorphism with transplanted kidney function. Ann Transplant. 2011;16:63–68
- Qu HQ, Bradfield JP, Grant SF, et al. Type I Diabetes Genetics Consortium. Remapping the type I diabetes association of the CTLA4 locus. Genes Immun. 2009;10:27–32
- Ladner MB, Bottini N, Valdes AM, et al. Association of the single nucleotide polymorphism C1858T of the PTPN22 gene with type 1 diabetes. Hum Immunol. 2005;66:60–64
- Mack R, Chowdary D, Samaan P, Podolak I, Dermody J. Prevalence of CTLA-4 polymorphism A49G in Ashkenazi Jews. Genet Test. 2001;5:269–271
- Kłoda K, Domanski L, Pawlik A, et al. Effect of the ICAM1 and VCAM1 gene polymorphisms on delayed graft function and acute kidney allograft rejection. Ann Transplant. 2010;15:15–20
- Domański L, Kłoda K, Pawlik A, et al. Correlation between ICAM1 and VCAM1 gene polymorphisms and histopathological changes in kidney allograft biopsies. Arch Med Sci. 2012. DOI: 10.5114/aoms.2012.29218
- Lahiri DK, Bye S, Nurnberger JI Jr. A non-organic and non-enzymatic extraction method gives higher yields of genomic DNA from whole-blood samples that do nine other methods tested. J Biochem Biophys Meth. 1992;25:193–205
- Tivol EA, Borriello F, Schweitzer AN, et al. Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4. Immunity. 1995;3:541–547
- Kearney ER, Walunas TL, Karr RW, et al. Antigen-dependent clonal expansion of a trace population of antigen-specific CD4+ T cells in vivo is dependent on CD28 costimulation and inhibited by CTLA-4. J Immunol. 1995;155:1032–1036
- Thompson CB, Allison JP. The emerging role of CTLA-4 as an immune attenuator. Immunity. 1997;7:445–450
- Wiśniewski A, Kusztal M, Magott-Procelewska M, et al. Possible association of cytotoxic T-lymphocyte antigen 4 gene promoter single nucleotide polymorphism with acute rejection of allogenic kidney transplant. Transplant Proc. 2006;38:56–58
- Kusztal M, Kościelska-Kasprzak K, Drulis-Fajdasz D, et al. The influence of CTLA-4 gene polymorphism on long-term kidney allograft function in Caucasian recipients. Transpl Immunol. 2010;23:121–124
- Magott-Procelewska M, Klinger M. New possibilities of therapeutic interventions in transplantation. Ann Transplant. 2004;9:19–24
- Chapman SJ, Khor CC, Vannberg FO, et al. PTPN22 and invasive bacterial disease. Nat Genet. 2006;38:499–500
- Azarian M, Busson M, Rocha V, et al. The PTPN22 R620W polymorphism is associated with severe bacterial infections after human leukocyte antigen geno-identical hematopoietic stem-cell transplantations. Transplantation. 2008;85:1859–1862
- Sfar I, Gorgi Y, Aouadi H, et al. The PTPN22 C1858T (R620W) functional polymorphism in kidney transplantation. Transplant Proc. 2009;41:657–659
- Sfar I, Ben Aleya W, Mouelhi L, et al. Lymphoid tyrosine phosphatase R620W variant and inflammatory bowel disease in Tunisia. World J Gastroenterol. 2010;16:479–483
- Lee YH, Rho YH, Choi SJ, et al. The PTPN22 C1858T functional polymorphism and autoimmune diseases—a meta-analysis. Rheumatology. 2007;46:49–56
- Gourh P, Tan FK, Assassi S, et al. Association of the PTPN22 R620W polymorphism with anti-topoisomerase I- and anticentromere antibody-positive systemic sclerosis. Arthritis Rheum. 2006;54:3945–3953
- Totaro MC, Tolusso B, Napolioni V, et al. PTPN22 1858C>T polymorphism distribution in Europe and association with rheumatoid arthritis: case-control study and meta-analysis. PLoS One. 2011;6:e24292
- Torres-Carrillo N, Ontiveros-Mercado H, Torres-Carrillo NM, et al. The -319C/+49G/CT60G haplotype of CTLA-4 gene confers susceptibility to rheumatoid arthritis in Mexican population. Cell Biochem Biophys. 2013 May 24 [Epub ahead of print]
- Prasad P, Kumar A, Gupta R, et al. Caucasian and Asian specific rheumatoid arthritis risk loci reveal limited replication and apparent allelic heterogeneity in north Indians. PLoS One. 2012;7:e31584