REFERENCES
- Firestein, G.S. (2003). Evolving concepts of rheumatoid arthritis. Nature 423:356–361.
- Pap, T., Muller Launder, Gary R.E. ,. (2000). Fibroblast biology: Role of synovial fibroblasts in pathogenesis of rheumatoid arthritis. Arthritis Res 2:361–367.
- Baier, A., Meineckel, I., Gap, S., . (2003). Apoptosis in rheumatoid arthritis. Cur Op in Rheumatol 15:274–2799.
- Liu, Hongtao, Wang, Yugang, Zhang, Yingmei. (1999). TRAP19, a novel apoptosis-related gene cloned from human leukemia cell line TF21, could enhance apoptosis of some tumor cells induced by growth factor withdrawl. Biochem Biophys Res Commun 254:203–210.
- Chen, Yingyu, Zhang, Yingmei, Sun, Yonghua. (2000). Preparation and identification of monoclonal antibody for human apoptosis-related protein TRAP19. Acta Acdemiate Medicinae Sinecae 22:502–504.
- Chen, Zhankun, Lv, Houshan, Wang, Ning. (2008). Expression of apoptotic gene PDCD5 in rheumatoid arthritis synovium. Chinese Journal of Rheumatology 12:36–39.
- Chen, Zhankun, Lv, Houshan, Wang, Ning. (2008). Upregulation of PDCD5 in rheumatoid arthritis fibroblast-like synoviocyte. Chinese Journal of Biochemistry and Molecular Biology 24:563–568.
- Arnett, F.C., Edworthy, S.M., Bloch, D.A. (1988). The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis. Arthritis Rheum 31:315–324.
- Fadok, V.A., Bratton, D.L., Frasch, S.C. (1998). The role of phosphatidylseine in recognition of apoptotic cells by phagocyte. Cell Death Differ 5:551–562.
- Smith, T.D., Waelde, C., Horwitz, A. (2002). Evaluation of the role of phosphatidylserine translocase activity in ABCA1-mediated lipid effhex. J Biol Chem 277: 17797–17803.
- Pope, R.M. (2002). Apoptosis as a threrapeutic tool in rheumatoid arthritis. Nat Rev Immunol 2:527–535.
- Li, Li, Chen, Yingyu, Zheng, Rui. (2000) TFAR19 expression in Jurket cell apoptosis induced by several modes. Chinese Journal of Experimental Hematology 8:81–84.
- Wang, Ying, Li, Xianting, Wang, Ling. (2004). An alternative form of paratosis-like cell death, triggered by TAJ/TROY and enhanced by PDCD5 overexpression. J Cell Sci 117:1525–1532.