Association of endothelial microparticle with NO, eNOS, ET-1, and fractional flow reserve in patients with coronary intermediate lesions

References

• Abid Hussein MN, Boing AN, Sturk A, et al. (2007). Inhibition of microparticle release triggers endothelial cell apoptosis and detachment. Thromb Haemost 98:1096–107
   PubMed Web of Science ®Google Scholar
• Bernal-Mizrachi L, Jy W, Jimenez JJ, et al. (2003). High levels of circulating endothelial microparticles in patients with acute coronary syndromes. Am Heart J 145:962–70
   PubMed Web of Science ®Google Scholar
• Brodsky SV, Zhang F, Nasjletti A, et al. (2004). Endothelium-derived microparticles impair endothelial function in vitro. Am J Physiol Heart Circ Physiol 286:H1910–5
   PubMed Web of Science ®Google Scholar
• Bulut D, Maier K, Bulut-Streich N, et al. (2008). Circulating endothelial microparticles correlate inversely with endothelial function in patients with ischemic left ventricular dysfunction. J Card Fail 14:336–40
   PubMed Web of Science ®Google Scholar
• Chironi GN, Boulanger CM, Simon A, et al. (2009). Endothelial microparticles in diseases. Cell Tissue Res 335:143–51
   PubMed Web of Science ®Google Scholar
• Chou YI, Kong J, Song R, et al. (2014). Correlations of platelet-derived microparticles with thromboxane B2, platelet-activating factor, endothelin-1, and neutrophil to lymphocyte ratio in patients with coronary intermediate lesions. Biomarkers 19:684–92
   PubMed Web of Science ®Google Scholar
• Hausmann D, Erbel R, Alibelli-Chemarin MJ, et al. (1995). The safety of intracoronary ultrasound. A multicenter survey of 2207 examinations. Circulation 91:623–30
   PubMed Web of Science ®Google Scholar
• Ikonomidis I, Athanassopoulos G, Lekakis J, et al. (2005). Myocardial ischemia induces interleukin-6 and tissue factor production in patients with coronary artery disease: a dobutamine stress echocardiography study. Circulation 112:3272–9
   PubMed Web of Science ®Google Scholar
• Kirma C, Akcakoyun M, Esen AM, et al. (2007). Relationship between endothelial function and coronary risk factors in patients with stable coronary artery disease. Circ J 71:698–702
   PubMed Web of Science ®Google Scholar
• Klinkner DB, Densmore JC, Kaul S, et al. (2006). Endothelium-derived microparticles inhibit human cardiac valve endothelial cell function. Shock 25:575–80
   PubMed Web of Science ®Google Scholar
• Koga H, Sugiyama S, Kugiyama K, et al. (2005). Elevated levels of VE-cadherin-positive endothelial microparticles in patients with type 2 diabetes mellitus and coronary artery disease. J Am Coll Cardiol 45:1622–30
   PubMed Web of Science ®Google Scholar
• Koupenova M, Johnston-Cox H, Ravid K. (2012). Regulation of atherosclerosis and associated risk factors by adenosine and adenosine receptors. Curr Atheroscler Rep 14:460–8
   PubMed Web of Science ®Google Scholar
• Kushner FG, Hand M, Smith SC Jr., et al. (2009). 2009 focused updates: ACC/AHA guidelines for the management of patients with ST-elevation myocardial infarction (updating the 2004 guideline and 2007 focused update) and ACC/AHA/SCAI guidelines on percutaneous coronary intervention (updating the 2005 guideline and 2007 focused update) a report of the American College of Cardiology Foundation/American Heart Association Task Force on Practice Guidelines. J Am Coll Cardiol 54:2205–41
   PubMed Web of Science ®Google Scholar
• Lei J, Vodovotz Y, Tzeng E, Billiar TR. (2013). Nitric oxide, a protective molecule in the cardiovascular system. Nitric Oxide 35:175–85
   PubMed Web of Science ®Google Scholar
• Lerman A. (2001). New insight and therapeutic strategies in cardiovascular disease and focus on endothelial target: endothelin-1 and angina. J Cardiovasc Pharmacol 38:S27–30
   PubMed Web of Science ®Google Scholar
• Leroyer AS, Ebrahimian TG, Cochain C, et al. (2009). Microparticles from ischemic muscle promotes postnatal vasculogenesis. Circulation 119:2808–17
   PubMed Web of Science ®Google Scholar
• Levine GN, Bates ER, Blankenship JC, et al. (2011). 2011 ACCF/AHA/SCAI guideline for percutaneous coronary intervention: a report of the American College of Cardiology Foundation/American Heart Association Task Force on practice guidelines and the Society for Cardiovascular Angiography and Interventions. Circulation 124:e574–651
   PubMed Web of Science ®Google Scholar
• Mallat Z, Benamer H, Hugel B, et al. (2000). Elevated levels of shed membrane microparticles with procoagulant potential in the peripheral circulating blood of patients with acute coronary syndromes. Circulation 101:841–3
   PubMed Web of Science ®Google Scholar
• Marenzi G, Lauri G, Assanelli E, et al. (2004). Contrast-induced nephropathy in patients undergoing primary angioplasty for acute myocardial infarction. J Am Coll Cardiol 44:1780–5
   PubMed Web of Science ®Google Scholar
• Markiewicz M, Richard E, Marks N, Ludwicka-Bradley A. (2013). Impact of endothelial microparticles on coagulation, inflammation, and angiogenesis in age-related vascular diseases. J Aging Res 2013:734509
   PubMedGoogle Scholar
• Mezentsev A, Merks RM, O'Riordan E, et al. (2005). Endothelial microparticles affect angiogenesis in vitro: role of oxidative stress. Am J Physiol Heart Circ Physiol 289:H1106–14
   PubMed Web of Science ®Google Scholar
• Namba M, Tanaka A, Shimada K, et al. (2007). Circulating platelet-derived microparticles are associated with atherothrombotic events: a marker for vulnerable blood. Arterioscler Thromb Vasc Biol 27:255–6
   PubMed Web of Science ®Google Scholar
• Nozaki T, Sugiyama S, Koga H, et al. (2009). Significance of a multiple biomarkers strategy including endothelial dysfunction to improve risk stratification for cardiovascular events in patients at high risk for coronary heart disease. J Am Coll Cardiol 54:601–8
   PubMed Web of Science ®Google Scholar
• Perez-Casal M, Downey C, Cutillas-Moreno B, et al. (2009). Microparticle-associated endothelial protein C receptor and the induction of cytoprotective and anti-inflammatory effects. Haematologica 94:387–94
   PubMed Web of Science ®Google Scholar
• Pernow J, Shemyakin A, Böhm F. (2012). New perspectives on endothelin-1 in atherosclerosis and diabetes mellitus. Life Sci 91:507–16
   PubMed Web of Science ®Google Scholar
• Poredos P. (2002). Endothelial dysfunction and cardiovascular disease. Pathophysiol Haemost Thromb 32:274–7
   PubMed Web of Science ®Google Scholar
• Robert S, Poncelet P, Lacroix R, et al. (2009). Standardization of platelet-derived microparticle counting using calibrated beads and a Cytomics FC500 routine flow cytometer: a first step towards multicenter studies? J Thromb Haemost 7:190–7
   PubMed Web of Science ®Google Scholar
• Sels JW, Elsenberg EH, Hoefer IE, et al. (2012). Fractional flow reserve is not associated with inflammatory markers in patients with stable coronary artery disease. PLoS One 7:e46356
   PubMed Web of Science ®Google Scholar
• Sessa WC. (2004). eNOS at a glance. J Cell Sci 117:2427–9
   PubMed Web of Science ®Google Scholar
• Tate DA, Bode AP, Nichols TC, et al. (1992). Platelet activation detected by platelet-derived microparticles in coronary sinus blood from patients with unstable coronary syndromes. Circulation 86:3193A
   Web of Science ®Google Scholar
• Tramontano AF, O'leary J, Black AD, et al. (2004). Statin decreases endothelial microparticle release from human coronary artery endothelial cells: implication for the Rho-kinase pathway. Biochem Biophys Res Commun 320:34–8
   PubMed Web of Science ®Google Scholar
• Tual-Chalot S, Guibert C, Muller B, et al. (2010). Circulating microparticles from pulmonary hypertensive rats induce endothelial dysfunction. Am J Respir Crit Care Med 182:261–8
   PubMed Web of Science ®Google Scholar
• van der Voort PH, van Hagen E, Hendrix G, et al. (1996). Comparison of intravenous adenosine to intracoronary papaverine for calculation of pressure-derived fractional flow reserve. Cathet Cardiovasc Diagn 39:120–5
   PubMedGoogle Scholar
• Vilahur G, Hernandez-Vera R, Molins B, et al. (2009). Short-term myocardial ischemia induces cardiac modified C-reactive protein expression and proinflammatory gene (cyclo-oxygenase-2, monocyte chemoattractant protein-1, and tissue factor) upregulation in peripheral blood mononuclear cells. J Thromb Haemost 7:485–93
   PubMed Web of Science ®Google Scholar