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Research Article
Host surface ectonucleotidase-CD73 and the opportunistic pathogen, Porphyromonas gingivalis, cross-modulation underlies a new homeostatic mechanism for chronic bacterial survival in human epithelial cells
Jaden S. Leea Department of Oral Health Sciences, Medical University of South Carolina, Charleston, SC, USACorrespondence[email protected]
https://orcid.org/0000-0001-6937-9416View further author information
https://orcid.org/0000-0001-6937-9416View further author information, Nityananda Chowdhurya Department of Oral Health Sciences, Medical University of South Carolina, Charleston, SC, USAhttps://orcid.org/0000-0002-8417-909XView further author information
https://orcid.org/0000-0002-8417-909XView further author information, JoAnn S. Robertsa Department of Oral Health Sciences, Medical University of South Carolina, Charleston, SC, USAhttps://orcid.org/0000-0003-2452-8750View further author information
https://orcid.org/0000-0003-2452-8750View further author information & Özlem Yilmaza Department of Oral Health Sciences, Medical University of South Carolina, Charleston, SC, USA;b Department of Microbiology and Immunology, Medical University of South Carolina, Charleston, SC, USACorrespondence[email protected]
https://orcid.org/0000-0003-3487-7217View further author information
https://orcid.org/0000-0003-3487-7217View further author informationRelated Research Data
Host surface ectonucleotidase-CD73 and the opportunistic pathogen, Porphyromonas gingivalis, cross-modulation underlies a new homeostatic mechanism for chronic bacterial survival in human epithelial cells
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Taylor & Francis
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