Supplemental material
Reprints and Corporate Permissions
Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?
To request a reprint or corporate permissions for this article, please click on the relevant link below:
Academic Permissions
Please note: Selecting permissions does not provide access to the full text of the article, please see our help page How do I view content?
Obtain permissions instantly via Rightslink by clicking on the button below:
If you are unable to obtain permissions via Rightslink, please complete and submit this Permissions form. For more information, please visit our Permissions help page.
Related Research Data
Very low-level heteroplasmy mtDNA variations are inherited in humans.
Source:
Elsevier BV
Understanding the Warburg Effect: The Metabolic Requirements of Cell Proliferation
Source:
American Association for the Advancement of Science (AAAS)
Use of stereotypical mutational motifs to define resolution limits for the ultra-deep resequencing of mitochondrial DNA.
Source:
Apollo - University of Cambridge Repository
A hybrid G-quadruplex structure formed between RNA and DNA explains the extraordinary stability of the mitochondrial R-loop
Source:
Oxford University Press (OUP)
The mitochondrial transcriptome of the anglerfish Lophius piscatorius
Source:
Springer Science and Business Media LLC
Mitochondrial mutations in cancer.
Source:
Springer Science and Business Media LLC
Down-regulation of the antisense mitochondrial non-coding RNAs (ncRNAs) is a unique vulnerability of cancer cells and a potential target for cancer therapy.
Source:
Amer Soc Biochemistry Molecular Biology Inc
Spectrum of somatic mitochondrial mutations in five cancers
Source:
Proceedings of the National Academy of Sciences
Enhanced glycolysis induced by mtDNA mutations does not regulate metastasis.
Source:
Federation of European Biochemical Societies. Published by Elsevier B.V.
Tight control of respiration by NADH dehydrogenase ND5 subunit gene expression in mouse mitochondria.
Source:
American Society for Microbiology
Humanin peptide suppresses apoptosis by interfering with Bax activation
Source:
Springer Science and Business Media LLC
Mitochondrial DNA genetics and the heteroplasmy conundrum in evolution and disease
Source:
Cold Spring Harbor Laboratory
Expanding the Coding Potential of Vertebrate Mitochondrial Genomes: Lesson Learned from the Atlantic Cod
Source:
IntechOpen
Mitochondria and cancer: a growing role in apoptosis, cancer cell metabolism and dedifferentiation
Source:
Springer Netherlands
Performance Comparison and Data Analysis Strategies for MicroRNA Profiling in Cancer Research
Source:
Springer International Publishing
A position 12-activated H-ras oncogene in all HS578T mammary carcinosarcoma cells but not normal mammary cells of the same patient
Source:
Proceedings of the National Academy of Sciences
Measurement of mitochondrial DNA copy number.
Source:
Humana Press
Long noncoding RNAs are generated from the mitochondrial genome and regulated by nuclear-encoded proteins
Source:
Cold Spring Harbor Laboratory
Mitochondrial dynamics and inheritance during cell division, development and disease.
Source:
Nature Publishing Group
Mitochondrial form and function
Source:
Springer Science and Business Media LLC
Distinct Small RNA Signatures in Extracellular Vesicles Derived from Breast Cancer Cell Lines
Source:
Public Library of Science (PLoS)
Mitogenome polymorphism in a single branch sample revealed by SOLiD deep sequencing of the Lophelia pertusa coral genome
Source:
Elsevier BV
Systematic Analysis of Small RNAs Associated with Human Mitochondria by Deep Sequencing: Detailed Analysis of Mitochondrial Associated miRNA
Source:
Public Library of Science (PLoS)
Two Syngeneic Cell Lines from Human Breast Tissue: The Aneuploid Mammary Epithelial (Hs578T) and the Diploid Myoepithelial (Hs578Bst) Cell Lines
Source:
Oxford University Press (OUP)
G-quadruplex structures in RNA stimulate mitochondrial transcription termination and primer formation
Source:
Proceedings of the National Academy of Sciences
Cancer-specific SNPs originate from low-level heteroplasmic variants in human mitochondrial genomes of a matched cell line pair
Source:
Taylor & Francis
Consistent treatment of length variants in the human mtDNA control region: a reappraisal
Source:
Springer Science and Business Media LLC
Reanalysis and revision of the Cambridge reference sequence for human mitochondrial DNA
Source:
Springer Science and Business Media LLC
Mitochondrial Dysfunction in Cancer
Source:
Frontiers Media SA
Mitochondrial genetic control of assembly and function of complex I in mammalian cells.
Source:
Springer Science and Business Media LLC
The awakening of an advanced malignant cancer: an insult to the mitochondrial genome.
Source:
Elsevier BV
Genome-wide screening for genetic changes in a matched pair of benign and prostate cancer cell lines using array CGH.
Source:
Springer Science and Business Media LLC
Linking provided by
