ERK2-dependent reactivation of Akt mediates the limited response of tumor cells with constitutive K-RAS activity to PI3K inhibition

Mahmoud Toulany Division of Radiobiology and Molecular Environmental Research; Department of Radiation Oncology; Eberhard Karls University Tuebingen; Tuebingen, Germany

Minjmaa Minjgee Division of Radiobiology and Molecular Environmental Research; Department of Radiation Oncology; Eberhard Karls University Tuebingen; Tuebingen, Germany

Mohammad Saki Division of Radiobiology and Molecular Environmental Research; Department of Radiation Oncology; Eberhard Karls University Tuebingen; Tuebingen, Germany

Marina Holler Division of Radiobiology and Molecular Environmental Research; Department of Radiation Oncology; Eberhard Karls University Tuebingen; Tuebingen, Germany

Friedegund Meier Division of Dermatologic Oncology; Department of Dermatology; University of Tuebingen; Tuebingen, Germany

Wolfgang Eicheler Department of Radiotherapy; University of Dresden; Dresden, Germany

H Peter Rodemann Division of Radiobiology and Molecular Environmental Research; Department of Radiation Oncology; Eberhard Karls University Tuebingen; Tuebingen, GermanyCorrespondence[email protected]

show all

ERK2-dependent reactivation of Akt mediates the limited response of tumor cells with constitutive K-RAS activity to PI3K inhibition

Additional material

Information for

Open access

Opportunities

Help and information

Your download is now in progress and you may close this window

Login or register to access this feature

ERK2-dependent reactivation of Akt mediates the limited response of tumor cells with constitutive K-RAS activity to PI3K inhibition

Additional material

Related research

To cite this article:

Download citation

Information for

Open access

Opportunities

Help and information

Keep up to date