Supplemental material
Epigenetics
Volume 14, 2019 - Issue 10
Research Paper
DNA methylation changes following DNA damage in prostate cancer cells
Laura P. SuttonSchool of Medicine, College of Health and Medicine, University of Tasmania, Hobart, Australia
https://orcid.org/0000-0002-1084-1941View further author information
https://orcid.org/0000-0002-1084-1941View further author information, Sarah A. JeffreysSchool of Medicine, College of Health and Medicine, University of Tasmania, Hobart, Australiahttps://orcid.org/0000-0002-3796-2268View further author information
https://orcid.org/0000-0002-3796-2268View further author information, Jessica L. PhillipsSchool of Medicine, College of Health and Medicine, University of Tasmania, Hobart, Australiahttps://orcid.org/0000-0002-5559-3072View further author information
https://orcid.org/0000-0002-5559-3072View further author information, Phillippa C. TaberlaySchool of Medicine, College of Health and Medicine, University of Tasmania, Hobart, Australiahttps://orcid.org/0000-0002-5298-8730View further author information
https://orcid.org/0000-0002-5298-8730View further author information, Adele F. HollowaySchool of Medicine, College of Health and Medicine, University of Tasmania, Hobart, Australiahttps://orcid.org/0000-0003-2213-1717View further author information
https://orcid.org/0000-0003-2213-1717View further author information, Mark AmbroseSchool of Medicine, College of Health and Medicine, University of Tasmania, Hobart, Australiahttps://orcid.org/0000-0002-8031-8240View further author information
https://orcid.org/0000-0002-8031-8240View further author information, Ji-Hoon E. JooColorectal Oncogenomics Group, Department of Clinical Pathology & University of Melbourne Centre for Cancer Research, The University of Melbourne, Parkville, Australiahttps://orcid.org/0000-0003-3331-5335View further author information
https://orcid.org/0000-0003-3331-5335View further author information, , Rachael BerrySchool of Medicine, College of Health and Medicine, University of Tasmania, Hobart, Australiahttps://orcid.org/0000-0001-7172-8830View further author information
https://orcid.org/0000-0001-7172-8830View further author information, Marketa SkalaDepartment of Radiation Oncology, Royal Hobart Hospital, Hobart, Australiahttps://orcid.org/0000-0002-1330-9811View further author information
https://orcid.org/0000-0002-1330-9811View further author information & Kate H. Brettingham-MooreSchool of Medicine, College of Health and Medicine, University of Tasmania, Hobart, AustraliaCorrespondence[email protected]
https://orcid.org/0000-0002-4304-4586View further author information
https://orcid.org/0000-0002-4304-4586View further author information show allRelated Research Data
Estimation of the radiation-induced DNA double-strand breaks number by considering cell cycle and absorbed dose per cell nucleus
Source:
Oxford University Press (OUP)
Genome-wide profiles of H2AX and γ-H2AX differentiate endogenous and exogenous DNA damage hotspots in human cells
Source:
Oxford University Press (OUP)
Rapid and transient recruitment of DNMT1 to DNA double-strand breaks is mediated by its interaction with multiple components of the DNA damage response machinery
Source:
Oxford University Press (OUP)
DNA methyltransferase inhibitor 5-aza-CdR enhances the radiosensitivity of gastric cancer cells
Source:
Wiley
5-Azacytidine, a DNA methyltransferase inhibitor, induces ATR-mediated DNA double-strand break responses, apoptosis, and synergistic cytotoxicity with doxorubicin and bortezomib against multiple myeloma cells
Source:
American Association for Cancer Research (AACR)
Recruitment of DNA methyltransferase I to DNA repair sites
Source:
Proceedings of the National Academy of Sciences
DNA damage and Repair Modify DNA methylation and Chromatin Domain of the Targeted Locus: Mechanism of allele methylation polymorphism
Source:
Springer Nature
Targeting the DNA Damage Response in Cancer
Source:
Elsevier BV
Chromatin Compaction Protects Genomic DNA from Radiation Damage
Source:
Public Library of Science (PLoS)
Quantitative DNA methylation predicts survival in adult acute myeloid leukemia
Source:
American Society of Hematology
Requirement of ATM-Dependent Phosphorylation of Brca1 in the DNA Damage Response to Double-Strand Breaks
Source:
American Association for the Advancement of Science (AAAS)
DNA double-strand breaks promote methylation of histone H3 on lysine 9 and transient formation of repressive chromatin.
Source:
Proceedings of the National Academy of Sciences
Enhancement of In vitro and In vivo Tumor Cell Radiosensitivity by the DNA Methylation Inhibitor Zebularine
Source:
American Association for Cancer Research (AACR)
Distinct mechanisms of regulation of the ITGA6 and ITGB4 genes by RUNX1 in myeloid cells
Source:
Wiley
Analysis of DNA methylation and gene expression in radiation-resistant head and neck tumors
Source:
Informa UK Limited
Heterochromatin and the DNA damage response: the need to relaxThis paper is one of a selection of papers in a Special Issue entitled 31st Annual International Asilomar Chromatin and Chromosomes Conference, and has undergone the Journal’s usual peer review process.
Source:
Canadian Science Publishing
5-Azacytidine Enhances the Radiosensitivity of CNE2 and SUNE1 Cells In Vitro and In Vivo Possibly by Altering DNA Methylation
Source:
Public Library of Science (PLoS)
Beyond Repair Foci: DNA Double-Strand Break Repair in Euchromatic and Heterochromatic Compartments Analyzed by Transmission Electron Microscopy
Source:
Public Library of Science (PLoS)
Underexpression of HOXA11 Is Associated with Treatment Resistance and Poor Prognosis in Glioblastoma
Source:
Korean Cancer Association
DNA Damage, Homology-Directed Repair, and DNA Methylation
Source:
Public Library of Science (PLoS)
DNMT (DNA methyltransferase) inhibitors radiosensitize human cancer cells by suppressing DNA repair activity
Source:
Springer Science and Business Media LLC
RNA-seq profiling of a radiation resistant and radiation sensitive prostate cancer cell line highlights opposing regulation of DNA repair and targets for radiosensitization
Source:
Springer Nature
5-azacytidine treatment reorganizes genomic histone modification patterns
Source:
Informa UK Limited
DNA Methylation Inhibitor 5-Aza-2'-Deoxycytidine Induces Reversible Genome-Wide DNA Damage That Is Distinctly Influenced by DNA Methyltransferases 1 and 3B
Source:
American Society for Microbiology
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