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Molecular and Cellular Biology Volume 23, 2003 - Issue 19
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Mammalian Genetic Models with Minimal or Complex Phenotypes

Disruption of the COP9 Signalosome Csn2 Subunit in Mice Causes Deficient Cell Proliferation, Accumulation of p53 and Cyclin E, and Early Embryonic Death

Karin Lykke-Andersen1 Department of Molecular, Cellular, and Developmental Biology, Yale University, New Haven, Connecticut06520-8104
,
Laura Schaefer2 Neuromuscular Biology & Disease Group, Boston Biomedical Research Institute, Watertown, Massachusetts02478
,
Suchithra Menon1 Department of Molecular, Cellular, and Developmental Biology, Yale University, New Haven, Connecticut06520-8104
,
Xing-Wang Deng1 Department of Molecular, Cellular, and Developmental Biology, Yale University, New Haven, Connecticut06520-8104
,
Jeffrey Boone Miller2 Neuromuscular Biology & Disease Group, Boston Biomedical Research Institute, Watertown, Massachusetts02478
&
Ning Wei1 Department of Molecular, Cellular, and Developmental Biology, Yale University, New Haven, Connecticut06520-8104Correspondence[email protected]
Pages 6790-6797 | Received 25 Apr 2003, Accepted 27 Jun 2003, Published online: 27 Mar 2023

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